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Barrow Neurological Institute is the world's largest neurological disease treatment and research institution, and is consistently ranked as one of the best neurosurgical training centers in the United States. [1] [2] Founded in 1962, the main campus is located at 350 W. Thomas Road in Phoenix, Arizona.
He is a graduate of Johns Hopkins School of Medicine and completed a residency at Barrow in 1997 which included a fellowship with Robert F. Spetzler. [2] He served as the vice chairman and chief of vascular neurosurgery at the UCSF for 20 years. He became the President and CEO of Barrow in 2017 when Speltzer retired. [3]
The Neurosurgery Residency Program at Barrow is the largest in the United States, training four residents per academic year, [2] for a total of 28 residents. In 2009 he became the Chief of the Spine Section at the Barrow Neurological Institute and was appointed the Volker K.H. Sonntag Chair in 2015.
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Robert F. Spetzler (born 1944) is a neurosurgeon and the J.N. Harber Chairman Emeritus of Neurological Surgery and director emeritus of the Barrow Neurological Institute in Phoenix, Arizona. [1] He retired as an active neurosurgeon in July 2017. [2]
He is well known for the Sonntag-Dickman Fusion, a special method of fusing the upper cervical spine. [20] [21] He was as a Scientific Reviewer and Chairman of the Scientific Program Committees of the North American Spine Society (NASS) [22] [7] and the Joint Spine Section of the AANS and CNS (1996). [23] In 2015, Dickman retired from ...
Rigid spine syndrome is a genetic disorder, primarily caused by mutations in the SEPN1 gene. [7] [8] This gene provides instructions for making a protein called selenoprotein N, which plays a role in muscle function and development. Mutations in the SEPN1 gene can lead to abnormal muscle stiffness and rigidity, as well as other characteristic ...
The three categories treated for types of spinal cord deficiencies are massive fusion of the cervical spine (Type I), the fusion of 1 or 2 vertebrae (Type II), and the presence of thoracic and lumbar spine anomalies in association with type I or type II Klippel–Feil syndrome (Type III). [citation needed]