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Paragonimiasis is a food-borne parasitic disease caused by several species of lung flukes belonging to genus Paragonimus. [4] Infection is acquired by eating crustaceans such as crabs and crayfishes which host the infective forms called metacercariae, or by eating raw or undercooked meat of mammals harboring the metacercariae from crustaceans.
Paragonimus westermani (Japanese lung fluke or oriental lung fluke) is the most common species of lung fluke that infects humans, causing paragonimiasis. [2] Human infections are most common in eastern Asia and in South America. Paragonimiasis may present as a sub-acute to chronic inflammatory disease of the lung. It was discovered by Dutch ...
The most prominent species of Paragonimus in human medicine is Paragonimus westermani, an infectious lung fluke originating in eastern Asia. Worldwide, about nine species of Paragonimus are known to cause human paragonimiasis in which many of the species reside in East Asia, West Africa, and in North and South America.
Paragonimus kellicotti, the North American lung fluke, is a species of parasitic trematode in the genus Paragonimus.This species of Paragonimus has an intricate lifecycle, and although its name may suggest that it is only a health concern in North America, it is also prominent in Southeast Asia and China.
First described by Chinese parasitologist Hsin-Tao Chen (1904–1977) in 1935, after examining Cantonese rat specimens, [1] the nematode Angiostrongylus cantonensis was identified in the cerebrospinal fluid of a patient with eosinophilic meningitis by Nomura and Lim in Taiwan in 1944.
The symptom of chigger bites that you're most likely to notice is, of course, the bites. Chigger bites tend to be small red, raised pimple-like itchy bumps.
Sparganosis is a parasitic infection caused by the plerocercoid larvae of the genus Spirometra including S. mansoni, S. ranarum, S. mansonoides and S. erinacei. [1] [2] It was first described by Patrick Manson in 1882, [3] and the first human case was reported by Charles Wardell Stiles from Florida in 1908. [4]
Inside the second intermediate host, the larvae will burrow into the intestinal tract where they develop into plerocercoid larvae (the final larval stage). In the final larvae stage, S. erinaceieuropaei migrate to subcutaneous tissues and/or muscles. [3] [6] Finally, the second intermediate host is eaten by a definitive host.