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Tinea versicolor (also pityriasis versicolor) is a condition characterized by a skin eruption on the trunk and proximal extremities. [1] The majority of tinea versicolor is caused by the fungus Malassezia globosa , although Malassezia furfur is responsible for a small number of cases.
Dermatophytosis, also known as tinea and ringworm, is a fungal infection of the skin [2] (a dermatomycosis), that may affect skin, hair, and nails. [1] Typically it results in a red, itchy, scaly, circular rash. [1]
These investigations show that the Malassezia species causing most skin disease in humans, including the most common cause of dandruff and seborrhoeic dermatitis, is M. globosa (though M. restricta is also involved). [1] The skin rash of tinea versicolor (pityriasis versicolor) is also due to infection by this fungus.
This fungus causes skin rashes, also known as tinea, which can spread on the face, limbs, groin, and feet, according to the press release. But unlike the neat circles typical with other forms of ...
[3] [6] Superficial fungal infections include common tinea of the skin, such as tinea of the body, groin, hands, feet and beard, and yeast infections such as pityriasis versicolor. [7] Subcutaneous types include eumycetoma and chromoblastomycosis, which generally affect tissues in and beneath the skin.
Tinea is often called "ringworm" because the rash is circular, with a ring-like appearance. It is sometimes equated with dermatophytosis, and, while most conditions identified as "tinea" are members of the imperfect fungi that make up the dermatophytes, conditions such as tinea nigra and tinea versicolor are not caused by dermatophytes.
Early studies are showing that inadvertent exposure in the first trimester of pregnancy was not associated with birth defects, but at the moment it is not recommended to take a GLP-1 during pregnancy.
Malassezia furfur is a fungus that lives on the superficial layers of the dermis.It generally exists as a commensal organism forming a natural part of the human skin microbiota, but it can gain pathogenic capabilities when morphing from a yeast to a hyphal form during its life cycle, through unknown molecular changes. [2]