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The term atypical TN is broad and due to the complexity of the condition, there are considerable issues with defining the condition further. Some medical practitioners no longer make a distinction between facial neuralgia (a nominal condition of inflammation) versus facial neuropathy (direct physical damage to a nerve). [citation needed]
40% of the adult population suffers from a headache disorder. Learn more about the different types of migraines and their symptoms.
The exact cause is unknown, but believed to involve loss of the myelin of the trigeminal nerve. [5] This might occur due to nerve compression from a blood vessel as the nerve exits the brain stem, multiple sclerosis, stroke, or trauma. Less common causes include a tumor or arteriovenous malformation. It is a type of nerve pain.
Migraine (UK: / ˈ m iː ɡ r eɪ n /, US: / ˈ m aɪ-/) [1] [2] is a genetically-influenced complex neurological disorder characterized by episodes of moderate-to-severe headache, most often unilateral and generally associated with nausea and light and sound sensitivity.
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Symptoms typically appear gradually over 5 to 20 minutes and generally last less than 60 minutes, leading to the headache in classic migraine with aura, or resolving without consequence in acephalgic migraine. [3] For many sufferers, scintillating scotoma is first experienced as a prodrome to migraine, then without migraine later in life ...
Facial onset sensory and motor neuronopathy, often abbreviated FOSMN, is a rare disorder of the nervous system in which sensory and motor nerves of the face and limbs progressively degenerate over a period of months to years. This degenerative process, the cause of which is unknown, eventually results in sensory and motor symptoms — the ...
Other sensory experiences, including tingling, numbness, difficulty speaking, or temporary alterations in hearing or smell, may also occur during the aura phase. The pathophysiology of migraines with aura is multifaceted, involving a complex interplay of genetic predispositions, environmental influences, and intricate neurobiological mechanisms.