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Weber's syndrome, also known as midbrain stroke syndrome or superior alternating hemiplegia, is a form of stroke that affects the medial portion of the midbrain. It involves oculomotor fascicles in the interpeduncular cisterns and cerebral peduncle so it characterizes the presence of an ipsilateral lower motor neuron type oculomotor nerve palsy and contralateral hemiparesis or hemiplegia.
Hemiparesis, also called unilateral paresis, is the weakness of one entire side of the body (hemi-means "half"). Hemiplegia, in its most severe form, is the complete paralysis of one entire side of the body.
Alternating hemiplegia (also known as crossed hemiplegia) is a form of hemiplegia that has an ipsilateral cranial nerve palsies and contralateral hemiplegia or hemiparesis of extremities of the body. The disorder is characterized by recurrent episodes of paralysis on one side of the body. [ 1 ]
Symptoms of cerebral infarction can help determine which parts of the brain are affected. If the infarct is located in the primary motor cortex , contralateral hemiparesis is said to occur. With brainstem localization, brainstem syndromes are typical: Wallenberg's syndrome , Weber's syndrome , Millard–Gubler syndrome , Benedikt syndrome or ...
Medial medullary syndrome, also known as inferior alternating syndrome, hypoglossal alternating hemiplegia, lower alternating hemiplegia, [1] or Dejerine syndrome, [2] is a type of alternating hemiplegia characterized by a set of clinical features resulting from occlusion of the anterior spinal artery.
Contralateral spastic hemiparesis: Medial lemniscus: Contralateral PCML (aka DCML) pathway loss (tactile, vibration, and stereognosis) Abducens nerve: Strabismus (ipsilateral lateral rectus muscle paralysis - the affected eye looks down and towards the nose). Abducens nerve lesion localizes the lesion to inferior pons.
Posterior cerebral artery is #6, and midbrain is behind it. Claude's syndrome is caused by midbrain infarction as a result of occlusion of a branch of the posterior cerebral artery. [2] This lesion is usually a unilateral infarction of the red nucleus and cerebellar peduncle, affecting several structures in the midbrain including:
Paroxysmal symptoms include tonic, tonic–clonic, or myoclonic limb movements, [8] dystonic posturing, choreoathetosis, ocular nystagmus, and various other ocular motor abnormalities. [1] [6] Almost half of all people have dystonic symptoms prior to experiencing hemiplegia. [4] These symptoms generally begin before 8 months of age. [8]