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Past studies link the consumption of fructose or high-fructose corn syrup to an increased risk for Alzheimer’s disease, type 2 diabetes, fatty liver disease, kidney disease, and cancer.
People with MASH cirrhosis have an increased risk of liver cancer. The rate of liver cancer associated with MASH increased fourfold between 2002 and 2012 in the US, which is more than any other cause of liver cancer. MASLD constitutes the third most common risk factor for liver cancer. [130] Cirrhosis is found in only about 50% of people with ...
Blocking this enzyme in prostate cancer cells grown in the lab stalled growth and triggered a catastrophic build-up of free-radicals, suggesting that it could be a suitable drug target. Importantly, this route to energy production is common to many different types of cancer, suggesting that drugs to target it could potentially be used to treat ...
Coenzyme Q cytochrome c reductase deficiency, Deficiency in enzymes of fat oxidation, Fructose intolerance, Galactosemia, Glycogen debranching deficiency, Hypoketonemic hypoglycemia, Ketotic hypoglycemia of infancy, Mcquarrie type infantile idiopathic hypoglycemia, Organic acidemia, Phosphoenolpyruvate carboxykinase (PEPCK) deficiency, Urea ...
Soda and processed-food manufacturers have long insisted that all sugars are essentially the same. Yet, simultaneously they're delicately backing away from high-fructose corn syrup (HFCS) as one ...
The absence of fructokinase results in the inability to phosphorylate fructose to fructose-1-phosphate within the cell. As a result, fructose is neither trapped within the cell nor directed toward its metabolism. [11] Free fructose concentrations in the liver increase and fructose is free to leave the cell and enter plasma.
Patel has also published research on genetic tests for cancer, [10] prostate cancer treatments, [11] fructose and liver health, [12] self-measured monitoring of blood pressure for hypertension, [13] and other clinical and methodological topics. [14]
This condition is caused by a deficiency of fructokinase in the liver. Affected individuals usually display a large blood fructose concentration after the ingestion of fructose, sucrose or sorbitol. [9] The disease is mainly characterized by the detection of the abnormal excretion of fructose in the urine through a urinalysis.