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Social stress can lead to adverse health outcomes by chronically activating the HPA axis or disrupting the HPA system. [95] There are a number of studies that link social stress and indications of a disrupted HPA axis; for instance, monkey infants neglected by their mothers show prolonged cortisol responses following a challenging event. [103]
Statistically, the direct effect hypothesis holds that there is no interaction between stress and social support, [21] meaning the same beneficial effects will be observed notwithstanding of the level of stress. Conversely, according to the social buffering hypothesis, the magnitude of the beneficial effect from social support is larger when ...
Minority stress theory extends the social causation hypothesis by suggesting that social situations do not lead directly to poor health for minority individuals, but that difficult social situations cause stress for minority individuals, which accrues over time, resulting in long-term health deficits. [22] [1] [23]
A stressor is a chemical or biological agent, environmental condition, external stimulus or an event seen as causing stress to an organism. [1] Psychologically speaking, a stressor can be events or environments that individuals might consider demanding, challenging, and/or threatening individual safety.
Transgenerational stress in humans, as in animal models, induces effects influencing social behavior, reproductive success, cognitive ability, and stress response. [3] Similar to animal models, human studies have investigated the role of epigenetics and transgenerational inheritance molecularly as it relates to the HPA system.
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Researchers have long been interested in how an individual's level and types of social support impact the effect of stress on their health. Studies consistently show that social support can protect against physical and mental consequences of stress. [80] [81] This can occur through a variety of mechanisms. One model, known as the "direct ...
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