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  2. Your Gout Guide: From Symptoms to Treatment - AOL

    www.aol.com/gout-guide-symptoms-treatment...

    If you have untreated gout for a long time, something called tophi can develop. Tophi is when uric acid crystals around the joints form larger, hard deposits. It can lead to pain, soft tissue ...

  3. Tophus - Wikipedia

    en.wikipedia.org/wiki/Tophus

    A tophus (Latin: "stone", pl.: tophi) is a deposit of monosodium urate crystals, in people with longstanding high levels of uric acid (urate) in the blood, a condition known as hyperuricemia. Tophi are pathognomonic for the disease gout. Most people with tophi have had previous attacks of acute arthritis, eventually leading to the formation of ...

  4. Gout - Wikipedia

    en.wikipedia.org/wiki/Gout

    Gout presenting as slight redness in the metatarsophalangeal joint of the big toe. Gout can present in several ways, although the most common is a recurrent attack of acute inflammatory arthritis (a red, tender, hot, swollen joint). [4] The metatarsophalangeal joint at the base of the big toe is affected most often, accounting for half of cases ...

  5. Wikipedia:VideoWiki/Gout - Wikipedia

    en.wikipedia.org/wiki/Wikipedia:VideoWiki/Gout

    Gout is a form of inflammatory arthritis characterized by recurrent attacks of a single red, ... It may also result in tophi, kidney stones, or urate nephropathy. [3 ...

  6. Pegloticase - Wikipedia

    en.wikipedia.org/wiki/Pegloticase

    Pegloticase (trade name Krystexxa) is a medication for the treatment of severe, treatment-refractory, chronic gout. It is a third line treatment in those in whom other treatments are not effective or are not tolerated. [2] The drug is administered by infusion intravenously. Since October 2023, Amgen Inc. has been the marketer of pegloticase in ...

  7. Probenecid - Wikipedia

    en.wikipedia.org/wiki/Probenecid

    In gout, probenecid competitively inhibits the reabsorption of uric acid through the organic anion transporter (OAT) at the proximal tubules. This leads to preferential reabsorption of probenecid back into plasma and excretion of uric acid in urine, [ 6 ] thus reducing blood uric acid levels and reducing its deposition in various tissues.

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