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The condition can cause localized pain, discoloration (paleness), and sensations of cold and/or numbness. When exposed to cold temperatures, the blood supply to the fingers or toes, and in some cases the nose or earlobes, is markedly reduced; the skin turns pale or white (called pallor ) and becomes cold and numb.
Pain, weakness, loss of muscle at the base of the thumb, swelling, paleness, bluish coloration [1] [2] Usual onset: 20 to 50 years of age [1] Types: Neurogenic, venous, arterial [1] Causes: Compression of the nerves, arteries, or veins in the superior thoracic aperture (thoracic outlet), the passageway from the lower neck to the armpit [1] Risk ...
It can cause symptoms such as: Chest pain, pressure, or tightness. Pain that spreads to your shoulder, arm, neck, or back. Cold sweats. ... sudden bleeding from a ruptured aneurysm. A ruptured ...
Shortness of breath is the most common symptom, followed by face or arm swelling. [5] Following are frequent symptoms: Difficulty breathing [6] Headache [6] Facial swelling [6] Venous distention in the neck and distended veins in the upper chest and arms [6] Migraines (especially if unusual to normal) Large decrease in lung capacity
Ischaemia would then result in the decrease in oxygen supply to the heart, hence weaken the heart. It causes a vicious cycle as a weakened heart contraction would decrease blood supply to the body, including cardiac muscle (muscle of heart). Moreover, if the plaque suddenly ruptures in the heart, it will cause a heart attack. [citation needed]
Anti-glomerular basement membrane (anti-GBM) vasculitis patients usually report sudden onset of anuria or oliguria. Typically, hematuria or tea-colored urine are noticed. [3] Many cases of cryoglobulinemia vasculitis are asymptomatic. [27] Hyperviscosity and/or thrombosis are the principal signs and symptoms of type I cryoglobulinemia.
It is differentiated from secondary causes of upper extremity thrombosis caused by intravascular catheters. [4] Paget–Schroetter syndrome was described once for a viola player who suddenly increased practice time 10-fold, creating enough repetitive pressure against the brachiocephalic and external jugular veins to cause thrombosis.
The patterns of draining veins from the fistula determines the risk of DAVF rupture. Increased pressure within the dural venous sinuses causes backpressure into the cortical veins, thus making cortical veins more prone to rupture. The risk of hemorrhage is graded by Cognard and Borden grading systems. These grading systems are based upon the ...