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White matter disease is strongly linked to cardiovascular disease risk factors, and researchers believe that white matter disease is a biomarker (medical sign) of the lifelong risk of stroke, dementia and disability.
The life expectancy of a person with white matter disease depends on many factors, including the specific type, the rate at which it progresses, and the complications it causes. Research has...
The life expectancy after a diagnosis of white matter disease depends on the speed it progresses and the severity of any other conditions it may cause, like stroke and dementia.
Age-related white matter disease is progressive, meaning it can get worse. But you can take steps to stop it from spreading.
Over the years it has become increasingly clear that the presence and extent of WMH is a radiographic marker of small cerebral vessel disease and an important predictor of the life-long risk of stroke, cognitive impairment, and functional disability.
White matter disease includes various conditions that affect white matter areas of the central nervous system. Some disorders can also affect other parts of the nervous system. Learn the types, causes, and treatments.
The outlook for individuals with PML depends on the severity of the underlying disease and treatment received. In general, PML has a mortality rate of 30 to 50 percent in the first few months following diagnosis. Those who survive the disease may be left with severe neurological disabilities.
Learn why white matter disease is common in aging, & how it's linked to cognitive decline, balance problems, vascular dementia, stroke and more.
Incidental findings of WMH on brain MRI may prompt screening for risk factors for stroke, cardiovascular events, and other chronic diseases. White matter hyperintensities may be used as a marker to evaluate the extent of small-vessel disease and thus predict long-term survival.
In this chapter, we specifically focus on WMH of vascular origin and explore white matter development, plasticity, and enduring processes of myelination across the health span in the context of experimental and human data, and compare and contrast resilient brain white matter propensity to a diseased white matter state.