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  2. Thrombocythemia - Wikipedia

    en.wikipedia.org/wiki/Thrombocythemia

    Other causes of reactive thrombocythemia include: post surgery, iron deficiency, drugs, and rebound effect after bone marrow suppression. [8] Research suggests that thrombocytosis can also occur after physical exercise, and is triggered by hemoconcentration and the release of platelets from the liver, lungs and spleen. [3] [9]

  3. Thrombopoietin - Wikipedia

    en.wikipedia.org/wiki/Thrombopoietin

    Thrombopoietin is a glycoprotein hormone produced by the liver and kidney which regulates the production of platelets. It stimulates the production and differentiation of megakaryocytes, the bone marrow cells that bud off large numbers of platelets. [5] Megakaryocytopoiesis is the cellular development process that leads to platelet production.

  4. Platelet-activating factor - Wikipedia

    en.wikipedia.org/wiki/Platelet-activating_factor

    The pathway to apoptosis can be inhibited by negative feedback from PAF acetylhydrolase (PAF-AH), an enzyme that catabolizes platelet-activating factor. It is an important mediator of bronchoconstriction. It causes platelets to aggregate and blood vessels to dilate. Thus, it is important to the process of hemostasis.

  5. Essential thrombocythemia - Wikipedia

    en.wikipedia.org/wiki/Essential_thrombocythemia

    Platelets derived from the abnormal megakaryocytes are activated, which, along with the elevated platelet count, contributes to the likelihood of forming blood clots. [8] The increased possibility of bleeding when the platelet count is over 1 million is due to von Willebrand factor (vWF) sequestration by the increased mass of platelets, leaving ...

  6. Antiplatelet drug - Wikipedia

    en.wikipedia.org/wiki/Antiplatelet_drug

    Antiplatelet therapy with one or more of these drugs decreases the ability of blood clots to form by interfering with the platelet activation process in primary hemostasis. Antiplatelet drugs can reversibly or irreversibly inhibit the process involved in platelet activation resulting in decreased tendency of platelets to adhere to one another ...

  7. Osteoarthritis - Wikipedia

    en.wikipedia.org/wiki/Osteoarthritis

    Knee pain relief of 50% or more following genicular RFA may last from several months to two years, [128] [131] and can be repeated by the same outpatient procedure when pain recurs. [128] Injection of phenol may be used as a neurolytic treatment of sensory knee nerves to relieve chronic pain from knee osteoarthritis. [133] [134]

  8. Sticky platelet syndrome - Wikipedia

    en.wikipedia.org/wiki/Sticky_platelet_syndrome

    Treatment usually consists of a daily low dose (80–100 mg) of aspirin. [13] Anticoagulants (e.g. warfarin) or antiplatelets (clopidogrel) are often additionally prescribed following formation of a medically significant clot. Thrombelastography is more commonly being used to diagnose hypercoagulability and monitor anti-platelet therapy.

  9. Platelet storage pool deficiency - Wikipedia

    en.wikipedia.org/wiki/Platelet_storage_pool...

    Platelet storage pool deficiency is a family of clotting disorders characterized by deficient granules in platelets. Individuals with these disorders have too few or abnormally functioning alpha granules , delta granules , or both alpha and delta granules and are therefore unable to form effective clots, which leads to prolonged bleeding.

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