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The infection, in these cases, spreads to the peritonsillar area (peritonsillitis). This region comprises loose connective tissue and is hence susceptible to formation of an abscess. PTA can also occur de novo. Both aerobic and anaerobic bacteria can be causative. Commonly involved aerobic pathogens include Streptococcus, Staphylococcus and ...
Lemierre's syndrome occurs most often when a bacterial (e.g., Fusobacterium necrophorum) throat infection progresses to the formation of a peritonsillar abscess. Deep in the abscess, anaerobic bacteria can flourish. When the abscess wall ruptures internally, the drainage carrying bacteria seeps through the soft tissue and infects the nearby ...
F. necrophorum is responsible for 10% of acute sore throats, [4] 21% of recurrent sore throats [5] [6] and 23% of peritonsillar abscesses [7] with the remainder being caused by Group A streptococci or viruses.
Strep throat is a common bacterial infection in children. [2] It is the cause of 15–40% of sore throats among children [7] [13] and 5–15% among adults. [8] Cases are more common in late winter and early spring. [13] Potential complications include rheumatic fever and peritonsillar abscess. [1] [2]
[1] [2] Complications include peritonsillar abscess (quinsy). [1] [3] Tonsillitis is most commonly caused by a viral infection and about 5% to 40% of cases are caused by a bacterial infection. [1] [5] [6] When caused by the bacterium group A streptococcus, it is classed as streptococcal tonsillitis [11] also referred to as strep throat. [12]
F. necrophorum has been found as a common pathogen in the diagnostic of peritonsillar abscess and is more prevalent than other bacteria regarding this infection. It is also the most frequent leading cause associated with Lemierre Syndrome and is not proven to be a normal part of the human oral bacterium population. [8]
It is a fastidious, slow growing, human commensal bacillus, capable of acting as an opportunistic pathogen and causing abscesses in several anatomical sites, including the liver, lung, spleen, and submandibular region. [4] E. corrodens could independently cause serious infection in both immunocompetent and immunocompromised hosts. [5]
The differential diagnosis must be established with acute and chronic tonsillitis, tonsillar hypertrophy, peritonsillar abscesses, foreign bodies, phlebolites, ectopic bone or cartilage, lymph nodes, granulomatous lesions or calcification of the stylohyoid ligament in the context of Eagle syndrome (elongated styloid process).