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Follicle-stimulating hormone (FSH) is a gonadotropin, a glycoprotein polypeptide hormone. [1] FSH is synthesized and secreted by the gonadotropic cells of the anterior pituitary gland [ 2 ] and regulates the development, growth, pubertal maturation , and reproductive processes of the body.
FSH, luteinizing hormone. thyroid-stimulating hormone, and hCG are all known to affect Sertoli cell development and male reproductive health. FSH is required for Sertoli cell mitogen, which stimulates the expression of various cell markers. [7]
Follicle-stimulating hormone: FSH Peptide: anterior pituitary: gonadotrope: FSH receptor → cAMP: In female: stimulates maturation of Graafian follicles in ovary. In male: spermatogenesis, enhances production of androgen-binding protein by the Sertoli cells of the testes. 27 Galanin: GAL Peptide: central nervous system and gastrointestinal ...
In the male, the FSH receptor has been identified on the Sertoli cells that are critical for spermatogenesis. [12] The FSHR is expressed during the luteal phase in the secretory endometrium of the uterus. [13] FSH receptor is selectively expressed on the surface of the blood vessels of a wide range of carcinogenic tumors. [14]
Gonadotropins are glycoprotein hormones secreted by gonadotropic cells of the anterior pituitary of vertebrates. [1] [2] [3] This family includes the mammalian hormones follicle-stimulating hormone (FSH) and luteinizing hormone (LH), the placental/chorionic gonadotropins, human chorionic gonadotropin (hCG) and equine chorionic gonadotropin (eCG), [3] as well as at least two forms of fish ...
During puberty, a male's erect penis becomes capable of ejaculating semen and impregnating a female. [26] [27] A male's first ejaculation is an important milestone in his development. [28] On average, a male's first ejaculation occurs at age 13. [29] Ejaculation sometimes occurs during sleep; this phenomenon is known as a nocturnal emission. [25]
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For example, the male mutation of the GnRH coding gene could result in hypogonadotrophic hypogonadism. A mutation that cause a gain of function for LH receptor can result in a condition known as testotoxicosis, which cause puberty to occur between ages 2–3 years. Loss of function of LH receptors can cause male pseudohermaphroditism.