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Important clinical infections caused by Enterococcus include urinary tract infections (see Enterococcus faecalis), bacteremia, bacterial endocarditis, diverticulitis, meningitis, and spontaneous bacterial peritonitis. [4] [8] [9] Sensitive strains of these bacteria can be treated with ampicillin, penicillin and vancomycin. [10]
Enterococcus faecalis – formerly classified as part of the group D Streptococcus system – is a Gram-positive, commensal bacterium inhabiting the gastrointestinal tracts of humans. [ 1 ] [ 2 ] Like other species in the genus Enterococcus , E. faecalis is found in healthy humans and can be used as a probiotic.
Enterococcus casseliflavus is a species of commensal Gram-positive bacteria. Its name derived from the "flavus" the Latin word for yellow due to the bright yellow pigment that it produces. [2] This organism can be found in the gastrointestinal tract of humans [3] The most common form of E. casseliflavus infection is bacteremia. [4]
Bacteremia is the presence of bacteria in the bloodstream that are alive and capable of reproducing. It is a type of bloodstream infection. [36] Bacteremia is defined as either a primary or secondary process. In primary bacteremia, bacteria have been directly introduced into the bloodstream. [37] Injection drug use may lead to primary bacteremia.
Enterococcus faecium has been a leading cause of multi-drug resistant enterococcal infections over Enterococcus faecalis in the United States. Approximately 40% of medical intensive care units reportedly found that the majority, respectively 80% and 90.4%, of device-associated infections (namely, infections due to central lines, urinary drainage catheters, and ventilators) were due to ...
Vancomycin. Six different types of vancomycin resistance are shown by enterococcus: Van-A, Van-B, Van-C, Van-D, Van-E and Van-G. [4] The significance is that Van-A VRE is resistant to both vancomycin and teicoplanin, [5] Van-B VRE is resistant to vancomycin but susceptible to teicoplanin, [6] [7] and Van-C is only partly resistant to vancomycin.
The mechanism of subacute bacterial endocarditis could be due to malformed stenotic valves which, in the company of bacteremia, become infected via adhesion and subsequent colonization of the surface area. This causes an inflammatory response, with recruitment of matrix metalloproteinases and destruction of collagen. [8]
In patients, pathogenic strains were found in the sputum, blood, wounds, and stool. Enterobacter is associated with common nosocomial infections including respiratory, endocarditis, bacteremia, urinary tract infections, osteomyelitis, among others. [8] Enterobacter bacteremia presents as fever but can progress to SIRS and shock. [8]