Search results
Results from the WOW.Com Content Network
The ethnic variation may be explained by genetic factors or simply because dark skinned people have greater amount of melanin in the mucosa, making it appear darker (termed racial or physiologic pigmentation). This darker mucosa may make the edematous changes more noticeable, whereas in the mucosa of people with lighter skin types leukoedema ...
Due to this, dark pigmentation may be visible on the oral mucosa or skin. [10] Most common oral sites include: buccal mucosa, lips, gums, hard palate or tongue. Intraoral sites are usually seen as the first sign and they usually develop prior to the skin lesions. [11]
Smoker melanosis in a patient consuming 2 packs of cigarette per day. Smoking or the use of nicotine-containing drugs is the cause to Smoker's melanosis. [10] [11] Tar-components (benzopyrenes) are also known to stimulate melanocytes to melanin production, and other unknown toxic agents in tobacco may also be the cause.
Generally, diseases outlined within the ICD-10 codes L80-L81 within Chapter XII: Diseases of the skin and subcutaneous tissue should be included in this category. Disturbances of human pigmentation, either loss or reduction, may be related to loss of melanocytes or the inability of melanocytes to produce melanin or transport melanosomes correctly.
Amalgam tattoo usually occurs on the mandibular gingiva, often in an area in which an apicoectomy ("root-end filling") with amalgam was carried out. [3]: 138 After the gingiva, the alveolar mucosa and the buccal mucosa are the next most common sites, although any mucosal site in the mouth is possible. [1]
Oral leukoplakia is a potentially malignant disorder affecting the oral mucosa. It is defined as "essentially an oral mucosal white/gray lesion that cannot be considered as any other definable lesion." Oral leukoplakia is a gray patch or plaque that develops in the oral cavity and is strongly associated with smoking. [8]
a. Early lesions, blanching of the oral mucosa; b. Older lesions, vertical and circular palpable fibrous bands in and around the mouth or lips, resulting in a mottled, marble-like appearance of the buccal mucosa; Stage 3: Sequelae of oral submucous fibrosis a. Leukoplakia; b. Speech and hearing deficits
Peutz–Jeghers syndrome (often abbreviated PJS) is an autosomal dominant genetic disorder characterized by the development of benign hamartomatous polyps in the gastrointestinal tract and hyperpigmented macules on the lips and oral mucosa (). [2]