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Rather than a disease, tinnitus is a symptom that may result from a variety of underlying causes and may be generated at any level of the auditory system as well as outside that system. The most common causes are hearing damage, noise-induced hearing loss, or age-related hearing loss, known as presbycusis. [2]
Though the pathophysiology of tinnitus is not known, noise exposure can be a contributing factor, therefore tinnitus can be associated with hearing loss, generated by the cochlea and central nervous system (CNS). High frequency hearing loss causes a high pitched tinnitus and low frequency hearing loss causes a roaring tinnitus. [19] Noise ...
The brain is more vulnerable to oxidative stress than other organs, due to its low oxidative capacity. Because neurons are characterized as postmitotic cells, meaning that they live with accumulated damage over the years, accumulation of ROS is fatal. Thus, increased levels of ROS age neurons, which leads to accelerated neurodegenerative ...
Definitions of hyperacusis can vary significantly; it often revolves around damage to or dysfunction of the stapes bone, stapedius muscle or tensor tympani . It is often categorized into four subtypes: loudness, pain (also called noxacusis), annoyance, and fear. [1] [2] It can be a highly debilitating hearing disorder. [3]
BrainHQ rewires the brain so you can think faster, focus better, and remember more. ... such as your joints, blood cells and organs. Multiple sclerosis, which causes damage to nerve cells. Cancer ...
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The stereocilia (hair cells) of the inner ear can become subjected to bending from loud noises. Because they are not regeneratable in humans, any major damage or loss of these hair cells leads to permanent hearing impairment and other hearing-related diseases. [2] Outer hair cells serve as acoustic amplifiers for stimulation of the inner hair ...
The drug is understood to damage multiple regions of the cochlea, causing the death of outer hair cells, as well as damage to the spiral ganglion neurons and cells of the stria vascularis. [27] Long-term retention of cisplatin in the cochlea may contribute to the drug's cochleotoxic potential. [ 28 ]
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