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Radioisotope renography is a form of medical imaging of the kidneys that uses radiolabelling.A renogram, which may also be known as a MAG3 scan, allows a nuclear medicine physician or a radiologist to visualize the kidneys and learn more about how they are functioning. [1]
Parenchymal destruction: The renal tissue undergoes caseous necrosis, fibrosis, and calcification. Fibrosis and shrinkage : Progressive scarring results in a small, irregularly shaped kidney. Calcification : Deposition of calcium salts within the necrotic tissue leads to the characteristic dense appearance of the kidney on imaging.
The kidney is divided into parenchyma and renal sinus. The renal sinus is hyperechoic and is composed of calyces, the renal pelvis, fat and the major intrarenal vessels. In the normal kidney, the urinary collecting system in the renal sinus is not visible, but it creates a heteroechoic appearance with the interposed fat and vessels.
It may cause acute kidney injury. It is now more commonly used to describe diffuse, fine, renal parenchymal calcification in radiology. [2] It is caused by multiple different conditions and is determined by progressive kidney dysfunction. These outlines eventually come together to form a dense mass. [3]
Anatomic pathology relates to the processing, examination, and diagnosis of surgical specimens by a physician trained in pathological diagnosis. Clinical pathology involves the laboratory analysis of tissue samples and bodily fluids; procedures may include blood sample analysis, urinalysis, stool sample analysis, and analysis of spinal fluid ...
The parenchymal cells include myocytes, and many types of specialised cells. The cells are often attached to each other and also to their nearby epithelial cells mainly by gap junctions and hemidesmosomes. There is much variation in the types of cell in the parenchyma according to the species and anatomical regions.
FE Na can be useful in the evaluation of acute kidney failure in the context of low urine output. Low fractional excretion indicates sodium retention by the kidney, suggesting pathophysiology extrinsic to the urinary system such as volume depletion or decrease in effective circulating volume (e.g. low output heart failure).
Since Page kidney is a unilateral process, symptom presentation differs significantly depending on if patients have native kidneys or only one functioning kidney, such as renal transplant recipients. [2] In those with a normally functioning second kidney, the only symptom may be new-onset hypertension.