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Mineral dust airway disease is a general term used to describe complications due to inhaled mineral dust causing fibrosis and narrowing of primarily the respiratory bronchioles. [1] It is a part of a group of disorders known as pneumoconioses which is characterized by inhaled mineral dust and the effects on the lungs.
The first known U.S. workers' compensation claim for asbestos disease was in 1927. In 1930, the first reported autopsy of a person with asbestosis was conducted in the United States and later presented by a doctor at the Mayo Clinic, although in this case the exposure involved mining activities somewhere in South America. [40]
The signs and symptoms of asbestosis typically manifest after a significant amount of time has passed following asbestos exposure, often several decades under current conditions in the US. [11] The primary symptom of asbestosis is generally the slow onset of shortness of breath , especially with physical activity. [ 12 ]
Occupational dust exposure occurs when small particles are generated at the workplace through the disturbance/agitation of rock/mineral, dry grain, timber, fiber, or other material. When these small particles become suspended in the air, they can pose a risk to the health of those who breath in the contaminated air.
Symptoms arise 4 to 12 hours after exposure to an organic dust, and generally last from one to five days. Common generalised symptoms include fever over 38 °C, chills, myalgia and malaise. The most frequent respiratory symptoms are dyspnea and a dry cough, while a wheeze may be present less commonly.
Exposure to coal dust is the cause of coalworker's pneumoconiosis, also called "black lung disease", is an interstitial lung disease caused by long-term exposure (over 10 years) to coal dust. Symptoms include shortness of breath and lowered pulmonary function. It can be fatal when advanced.
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Silicosis that develops 5–10 years after first exposure to higher concentrations of silica dust. Symptoms and x-ray findings are similar to chronic simple silicosis, but occur earlier and tend to progress more rapidly. Patients with accelerated silicosis are at greater risk for complicated disease, including progressive massive fibrosis (PMF).