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[11] [18] This is sometimes accompanied by the development of a crazy paving pattern and interlobular septal thickening. [18] In many cases the most severe pulmonary CT abnormalities occurred within 2 weeks after symptoms began. [17] At this point, many individuals begin showing resolution of consolidation and GGOs as symptoms improve.
On CT, lipoid pneumonia appears as a "crazy paving" pattern, characterized by ground glass opacities with interspersed interlobular septal thickening. [ 8 ] Diagnosis
Pneumonitis may cause subpleural honeycombing, changing the shape of the air spaces in an image, which may be used to identify the respiratory disease. [7] The interlobular septa may also thicken and indicate pneumonitis when viewed on a scan. [7]
Predominantly subpleural and basal; Often heterogenous distribution, being occasionally diffuse, and may be asymmetrical; There may be superimposed CT features such as mild ground-glass opacity, reticular pattern and pulmonary ossification. Probable UIP pattern: [4] Predominantly subpleural and basal; Often heterogenous distribution
On radiological studies, diffuse lesions are visualized throughout the thoracic cavity using CT scans. The location of the lesions is mostly in the upper lobes of the lungs, usually in a lymphatic distribution. Thickening of the pleura and interlobular septal is also evident.
Pulmonary interstitial emphysema (PIE) is a collection of air inside the lungs but outside the normal air space of the alveoli, found as pneumatoses inside the connective tissue of the peribronchovascular sheaths, interlobular septa, and visceral pleura.
These are short parallel lines at the lung periphery. These lines represent interlobular septa, which are usually less than 1 cm in length and parallel to one another at right angles to the pleura. They are located peripherally in contact with the pleura, but are generally absent along fissural surfaces.
This type of alveolar damage can be attributed to nonconcentrated and nonlocalized alveoli damage, marked alveolar septal edema with inflammatory cell infiltration, fibroblast proliferation, occasional hyaline membranes, and thickening of the alveolar walls. The septa are lined with atypical, hyperplastic type II pneumocytes, thus leading to ...