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The cause of dysentery is usually the bacteria from genus Shigella, in which case it is known as shigellosis, or the amoeba Entamoeba histolytica; then it is called amoebiasis. [1] Other causes may include certain chemicals, other bacteria, other protozoa, or parasitic worms. [2] It may spread between people. [4]
It is sometimes listed as an explicit differential diagnosis of bacillary dysentery, as opposed to a cause. [6] Bacillary dysentery should not be confused with diarrhea caused by other bacterial infections. One characteristic of bacillary dysentery is blood in stool, [7] which is the result of invasion of the mucosa by the pathogen.
Shigellosis, known historically as dysentery, is an infection of the intestines caused by Shigella bacteria. [ 1 ] [ 3 ] Symptoms generally start one to two days after exposure and include diarrhea , fever , abdominal pain , and feeling the need to pass stools even when the bowels are empty. [ 1 ]
[3] [4] During infection, it typically causes dysentery. [5] Shigella is a leading cause of bacterial diarrhea worldwide, with 80–165 million annual cases (estimated) [6] and 74,000 to 600,000 deaths. [6] [7] It is one of the top four pathogens that cause moderate-to-severe diarrhea in African and South Asian children. [8]
The most commonly observed signs associated with Shigella dysentery include colitis, malnutrition, rectal prolapse, tenesmus, reactive arthritis, and central nervous system problems. Further, S. dysenteriae is associated with the development of hemolytic-uremic syndrome, which includes anemia, thrombocytopenia, and kidney failure.
Erythromycin can be used in children, and tetracycline in adults. Some studies show, however, that erythromycin rapidly eliminates Campylobacter from the stool without affecting the duration of illness. Nevertheless, children with dysentery due to C. jejuni benefit from early treatment with erythromycin. Treatment with antibiotics, therefore ...
A study found that trichrome staining, a traditional method for identification, had a sensitivity of 36% (9/25) when compared to stool culture. [11] An additional study found that the sensitivity of staining was 50% (2/4), and that the organism could be successfully cultured in stool specimens up to 12-hours old that were kept at room temperature.
This IcsA/N-WASP complex then activates the Actin-related protein (Arp) 2/3 Complex. Arp 2/3 Complex is the protein responsible for rapidly initiating actin polymerization and propelling the bacteria forward. [8] [2] [9] When S. flexneri reaches the adjoining membrane, it creates a protrusion into the neighboring cell's cytoplasm. The bacteria ...