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Attention deficit hyperactivity disorder; Other names: Formerly: Attention deficit disorder (ADD), hyperkinetic disorder (HD) [1]: ADHD arises from maldevelopment in brain regions such as the prefrontal cortex, basal ganglia and anterior cingulate cortex, which regulate the executive functions necessary for human self-regulation.
p53, also known as Tumor protein P53, cellular tumor antigen p53 (UniProt name), or transformation-related protein 53 (TRP53) is a regulatory protein that is often mutated in human cancers. The p53 proteins (originally thought to be, and often spoken of as, a single protein) are crucial in vertebrates , where they prevent cancer formation. [ 5 ]
The hypothesis notes that humans spent most of their evolutionary history in hunter-gatherer societies, and it argues that ADHD represents a lack of adaptation to farming societies. Hartmann first developed the idea as a mental model after his own son was diagnosed with ADHD, stating, "It's not hard science, and was never intended to be." [1]
Attention deficit hyperactivity disorder predominantly inattentive (ADHD-PI or ADHD-I), [3] is one of the three presentations of attention deficit hyperactivity disorder (ADHD). [4] In 1987–1994, there were no subtypes or presentations and thus it was not distinguished from hyperactive ADHD in the Diagnostic and Statistical Manual (DSM-III-R).
Parkinson's disease, attention deficit hyperactivity disorder (ADHD), substance use disorders , and restless legs syndrome (RLS) can be attributed to dysfunction in specific dopaminergic pathways. The dopamine neurons of the dopaminergic pathways synthesize and release the neurotransmitter dopamine.
Adult Attention Deficit Hyperactivity Disorder is the persistence of attention deficit hyperactivity disorder (ADHD) into adulthood. It is a neurodevelopmental disorder, meaning impairing symptoms must have been present in childhood, except for when ADHD occurs after traumatic brain injury.
Hyperactivity has long been part of the human condition, although hyperactive behaviour has not always been seen as problematic. [1] [page needed]The terminology used to describe the symptoms of attention deficit hyperactivity disorder, or ADHD, has gone through many changes over history, including "minimal brain damage", "minimal brain dysfunction", "learning/behavioral disabilities" and ...
[9] p53 is also phosphorylated by the effector kinase CHK2. These phosphorylation events lead to stabilization and activation of p53 and subsequent transcription of numerous p53 target genes including CDK inhibitor p21 which lead to long-term cell-cycle arrest or even apoptosis. [15] ATM-mediated two-step response to DNA double strand breaks.
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