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Tums (stylized as TUMS) is an antacid made of sucrose (table sugar) and calcium carbonate (CaCO 3) manufactured by Haleon in St. Louis, Missouri, US. They are also available in a sugar-free version. It is an over-the-counter drug, available at many retail stores, including drug stores, grocery stores and mass merchandisers.
Nephrotoxicity is toxicity in the kidneys. It is a poisonous effect of some substances, both toxic chemicals and medications, on kidney function. [1] There are various forms, [2] and some drugs may affect kidney function in more than one way. Nephrotoxins are substances displaying nephrotoxicity.
This illustration demonstrates the normal kidney physiology, including the Proximal Convoluted Tubule (PCT), Loop of Henle, and Distal Convoluted Tubule (DCT). It also includes illustrations showing where some types of diuretics act, and what they do. Renal physiology (Latin renes, "kidneys") is the study of the physiology of the kidney.
The post How Tums Antacid Became America’s Most Trusted Brand appeared first on Reader's Digest. These tiny tablets have been providing relief to overfull tummies for nearly a century, earning ...
When the kidneys are damaged, potassium levels can become elevated. Elevated potassium levels can cause the heart to beat irregularly or even stop. Phosphorus: The kidneys also help to regulate phosphorus levels in the body. When the kidneys are damaged, they aren't able to remove excessive phosphorus, causing it to build up in blood.
But her nephrologist diagnosed her with stage 4 kidney failure, a precursor to chronic kidney disease (CKD), and she would eventually have to start regular dialysis.
In more extreme cases, experts said, holding pee can cause urine to back up into the kidneys, which can lead to infections, kidney damage or hydronephrosis. The latter is a condition in which the ...
Milk-alkali syndrome (MAS), also referred to as calcium-alkali syndrome, is the third most common cause of elevated blood calcium levels (hypercalcemia). [2] [3] Milk-alkali syndrome is characterized by hypercalcemia, metabolic alkalosis, and acute kidney injury.
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