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Congenital lobar emphysema (CLE), also known as congenital lobar overinflation and infantile lobar emphysema, [37] is a neonatal condition associated with enlarged air spaces in the lungs of newborn infants. It is diagnosed around the time of birth or in the first 6 months of life, occurring more often in boys than girls.
Congenital pulmonary airway malformation (CPAM), formerly known as congenital cystic adenomatoid malformation (CCAM), is a congenital disorder of the lung similar to bronchopulmonary sequestration. In CPAM, usually an entire lobe of lung is replaced by a non-working cystic piece of abnormal lung tissue.
In children, additional causes include measles, echinococcosis, inhalation of a foreign body, and certain congenital malformations (congenital pulmonary airway malformation and congenital lobar emphysema). [19] 11.5% of people with a spontaneous pneumothorax have a family member who has previously experienced a pneumothorax.
Pleural effusions can also develop, which are also seen with meconium aspiration but not with respiratory distress syndrome. [2] The lungs may also appear hyperinflated. [5] It is a diagnosis of exclusion as it is a benign condition that can have symptoms and signs similar to more serious syndromes, such as respiratory distress or meconium ...
Congenital pulmonary airway malformation, formerly called congenital cystic adenomatoid malformation, is a benign tumor the results in the formation of single or multiple cysts. [2] Pulmonary sequestration refers to abnormal lung tissue that gets its blood supply from the systemic circulation instead of the pulmonary circulation, like the rest ...
Low magnification micrograph of pneumatosis intestinalis in bowel wall.. Pneumoperitoneum (or peritoneal emphysema), air or gas in the abdominal cavity.The most common cause is a perforated abdominal viscus, generally a perforated peptic ulcer, although any part of the bowel may perforate from a benign ulcer, tumor or abdominal trauma.
Pulmonary interstitial emphysema (PIE) is a collection of air outside of the normal air space of the pulmonary alveoli, found instead inside the connective tissue of the peribronchovascular sheaths, interlobular septa, and visceral pleura. (This supportive tissue is called the pulmonary interstitium.)
The condition is marked by induration [2] (swelling or hardening of normally soft tissue) of a normally aerated lung. It is considered a radiologic sign . Consolidation occurs through accumulation of inflammatory cellular exudate in the alveoli and adjoining ducts.
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