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For children with MRSA-infected bone or joints, treatment is individualized and long-term. Neonates can develop neonatal pustulosis as a result of topical infection with MRSA. [4] Clindamycin is not approved for the treatment of MRSA infection, but it is still used in children for soft-tissue infections. [4]
Because of the high level of resistance to penicillins and because of the potential for MRSA to develop resistance to vancomycin, the U.S. Centers for Disease Control and Prevention has published guidelines [121] for the appropriate use of vancomycin. In situations where the incidence of MRSA infections is known to be high, the attending ...
Resistance in VRSA is conferred by the plasmid-mediated vanA gene and operon. [5] Although VRSA infections are uncommon, VRSA is often resistant to other types of antibiotics and a potential threat to public health because treatment options are limited. [6] VRSA is resistant to many of the standard drugs used to treat S. aureus infections ...
The Task Force for Combating Antibiotic-Resistant Bacteria developed The National Action Plan for Combating Antibiotic-Resistant Bacteria with the intent of providing a roadmap to guide the US in the antibiotic resistance challenge and with hopes of saving many lives. This plan outlines steps taken by the Federal government over the next five ...
Staphylococcus aureus. MRSA ST398 is a strain of the gram-positive bacterium Staphylococcus aureus, which belongs to the genus Staphylococcus. This genus covers a large group of gram-positive bacteria that are classified taxonomically in the family Staphylococcaceae, order Bacillales, class Bacilli, and phylum Firmicutes.
Since many R-factors contain F-plasmids, antibiotic resistance can be easily spread among a population of bacteria. [19] Also, R-factors can be taken up by "DNA pumps" in their membranes via transformation , [ 20 ] or less commonly through viral mediated transduction , [ 21 ] or via bacteriophage, although conjugation is the most common means ...
SCCmec, or staphylococcal cassette chromosome mec, is a mobile genetic element of Staphylococcus bacterial species. This genetic sequence includes the mecA gene coding for resistance to the antibiotic methicillin and is the only known way for Staphylococcus strains to spread the gene in the wild by horizontal gene transfer. [1]
S. aureus is a non-motile bacteria, and must rely on alternative forms of spreading. PSMs have been implicated in assisting with colony spreading. [12] PSMα 1-4 have been shown to help S. aureus colonies spread on agar plates. [12] However, δ-Toxin, which is another α-class PSM, does not play a role in colony spreading. [12]