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In up to 10% of cases there is a family history of the disorder. Family history is more common in narcolepsy with cataplexy. [1] There is a strong link with certain genetic variants, [27] which may make T-cells susceptible to react to the orexin-releasing neurons (autoimmunity) [30] after being stimulated by infection with H1N1 influenza. [12]
Cataplexy is the first symptom to appear in about 10% of cases of narcolepsy, [2] caused by an autoimmune destruction of hypothalamic neurons that produce the neuropeptide hypocretin (also called orexin), which regulates arousal and has a role in stabilization of the transition between wake and sleep states. [3]
A large percentage of people with narcolepsy have cataplexy as well, which along with excessive sleepiness disrupts a person’s ability to socialize, work at a job, drive a car or even have close ...
Cataplexy, on the other hand, is an involuntary loss of muscle tone during wakefulness. The mechanism of narcolepsy is unknown, though recent findings suggest that orexin neurons in the lateral and posterior hypothalamus may play a critical role in reinforcing wakefulness. [ 7 ]
Experts say cataplexy, which is a loss of muscle tone, often accompanies narcolepsy. In the video, Sarah was working on a traditional Japanese dance to send to a choreographer friend. "I realized ...
Primary vs. secondary (i.e. comorbid) insomnia has been reunited into a single disorder: chronic insomnia. Narcolepsy has been divided into narcolepsy type 1 and narcolepsy type 2. These two types are distinguished by the presence or absence of cataplexy and the cerebrospinal fluid hypocretin-1 level.
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