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The gastrocnemius muscle is prone to spasms, which are painful, involuntary contractions of the muscle that may last several minutes. [5] A severe ankle dorsiflexion force may result in a Medial Gastrocnemius Strain (MGS) injury of the muscle, commonly referred to as a "torn" or "strained" calf muscle, which is acutely painful and disabling. [6]
A burst cyst commonly causes calf pain, swelling and redness that may mimic thrombophlebitis. A specimen from a cadaver of a Baker's cyst in popliteal space Baker's cyst on axial MRI with communicating channel between the semimembranosus muscle and the medial head of the gastrocnemius muscle.
Sometimes, the SSV joins the common gastrocnemius vein before draining in the popliteal vein. [2] Sometimes, it does not make contact with the popliteal vein, but goes up to drain in the GSV at a variable level. [1] Instead of draining in the popliteal vein, it can merge with the Giacomini vein and drain in the GSV at the superior 1/3 of the thigh.
The popliteal vein may become trapped. [5] This reduces the flow of blood out of the leg, causing oedema, pain, and venous ulcers. [5] Entrapment is usually caused by gastrocnemius muscle. [5] Venography (using an x-ray) or magnetic resonance imaging can investigate it. [5] Surgery can be used to remove tissue creating pressure. [5]
The next step of identifying the POL femoral attachment is done by locating the gastrocnemius tubercle (2.6 mm distal and 3.1 mm anterior to the medial gastrocnemius tendon attachment on the femur). If the posteromedial capsule is not intact, the POL attachment site is located 7.7 mm distal and 2.9 mm anterior to the gastrocnemius tubercle.
Medial gastrocnemius perforator, draining into the gastrocnemius vein; Fibular perforators, usually two, one superior near the lateral aspect of the knee and one inferior at the lateral aspect of the ankle; When the valves of perforator veins become incompetent they can cause venous reflux when the muscles contract.
Biarticular muscles can also transfer mechanical power between distal and proximal joints, though the direction and magnitude of this transfer varies with anatomy, muscle activity level, and joint angles. This is a crucial consideration when analyzing an organism's movement using inverse dynamics.
The disease is characterized by slowly progressive weakness and wasting of both proximal muscles (located on or close to the torso) and distal muscles (close to hands or feet), most apparent in the finger flexors and knee extensors. [3] IBM is often confused with an entirely different class of diseases, called hereditary inclusion body ...