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This swinging of the heart causes electrical alternans seen on EKG. A pericardial effusion is fluid in the pericardial sac. When large enough, the pressure compresses the heart. This causes shock by preventing the heart from filling with blood. This is called cardiac tamponade. The chambers of the heart can collapse from this pressure.
A pericardial effusion with enough pressure to adversely affect heart function is called cardiac tamponade. [1] Pericardial effusions can cause cardiac tamponade in acute settings with fluid as little as 150mL. In chronic settings, however, fluid can accumulate anywhere up to 2L before an effusion causes cardiac tamponade.
Cardiac tamponade, also known as pericardial tamponade (/ ˌ t æ m. p ə ˈ n eɪ d / [4]), is a compression of the heart due to pericardial effusion (the build-up of pericardial fluid in the sac around the heart). [2] Onset may be rapid or gradual. [2]
Purulent Pericarditis; Echocardiogram showing pericardial effusion with signs of cardiac tamponade: Specialty: Cardiology: Symptoms: substernal chest pain (exacerbated supine and with breathing deeply), dyspnea, fever, rigors/chills, and cardiorespiratory signs (i.e., tachycardia, friction rub, pulsus paradoxus, pericardial effusion, cardiac tamponade, pleural effusion)
Pericardiocentesis can be used to diagnose and treat cardiac tamponade. [3] [4] Cardiac tamponade is a medical emergency in which excessive accumulation of fluid within the pericardium (pericardial effusion) creates increased pressure. [5] This prevents the heart from filling normally with blood.
The severity of hemorrhagic shock can be graded on a 1–4 scale on the physical signs. The shock index (heart rate divided by systolic blood pressure) is a stronger predictor of the impact of blood loss than heart rate and blood pressure alone. [11] This relationship has not been well established in pregnancy-related bleeding. [12]
Neurogenic shock can result from severe central nervous system damage (brain injury, cervical or high thoracic spinal cord). [1]In simple terms, the trauma causes a sudden loss of background SNS stimulation to the blood vessels.
The typical signs of post-pericardiotomy syndrome include fever, pleuritis (with possible pleural effusion), pericarditis (with possible pericardial effusion), occasional but rare pulmonary infiltrates, and fatigue. [1] [2] Cough, pleuritic or retrosternal chest pain, joint pain and decreased oxygen saturation can also be seen in some cases. [1]