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It can be detected through the use of a reflex hammer and is the opposite of hyperreflexia. [ citation needed ] Hyporeflexia is generally associated with a deficit in the lower motor neurons (at the alpha motor neurons from the spinal cord to a muscle), whereas hyperreflexia is often attributed to lesions in the upper motor neurons (along the ...
The Golgi tendon reflex is a response to extensive tension on a tendon. [7] It helps avoid strong muscle contractions which could tear the tendon from either the muscle or bone. [7] In sports, quick movements can damage the tendon before the reflex can occur. [7]
Woltman's sign (also called Woltman's sign of hypothyroidism or, in older references, myxedema reflex [1]) is a delayed relaxation phase of an elicited deep tendon reflex, usually tested in the Achilles tendon of the patient. Woltman's sign is named for Henry Woltman, an American neurologist. [2]
It is a type of stretch reflex that tests the function of the gastrocnemius muscle and the nerve that supplies it. A positive result would be the jerking of the foot towards its plantar surface. Being a deep tendon reflex, it is monosynaptic. It is also a stretch reflex. These are monosynaptic spinal segmental reflexes.
Glabellar reflex; Golgi tendon reflex; Hanger reflex - reflex of unclear purpose that causes the head to rotate to the right when the top sides of the head are under pressure, named because it can be easily activated with a coat hanger; Hering–Breuer reflex — is a reflex triggered to prevent over-inflation of the lung
The Golgi tendon reflex operates as a protective feedback mechanism to control the tension of an active muscle by causing relaxation before the tendon tension becomes high enough to cause damage. [7] First, as a load is placed on the muscle, the afferent neuron from the Golgi tendon organ fires into the central nervous system .
Harding ataxia is an autosomal recessive cerebellar ataxia originally described by Harding in 1981. [1] This form of cerebellar ataxia is similar to Friedreich ataxia including that it results in poor reflexes and balance, but differs in several ways, including the absence of diabetes mellitus, optic atrophy, cardiomyopathy, skeletal abnormalities, and the fact that tendon reflexes in the arms ...
This results in decreased deep tendon reflexes (hyporeflexia), and skeletal muscle weakness. [6] Other symptoms include cardiac arrhythmias (especially in those taking digoxin), fatigue, nausea, vomiting (emesis), loss of appetite, abdominal pain, & paralytic ileus.