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Low sodium intake level was a mean of <115 mmol (2645 mg), usual sodium intake was 115-215 mmol (2645–4945 mg), and a high sodium intake was >215 mmol (4945 mg), concluding: "Both low sodium intakes and high sodium intakes are associated with increased mortality, consistent with a U-shaped association between sodium intake and health outcomes".
“Zero dietary sodium intake may lead to clinically significant low blood sodium levels, which can cause severe muscle cramps, weakness, nausea and vomiting, seizures and coma, shock or even ...
Women are found to be more sodium sensitive than men; one possible explanation is based on the fact that women tend to consume more salt per unit weight, as women weigh less than men on average. [32] Several studies have shown that the increase in age is also associated with the occurrence of sodium sensitivity. [31]
For certain people with salt-sensitive blood pressure or diseases such as Ménière's disease, this extra intake may cause a negative effect on health. WHO guidelines [4] [5] state that adults should consume less than 2,000 mg of sodium/day (i.e. about 5 grams of traditional table salt), and at least 3,510 mg of potassium per day. [6]
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Swapping regular salt for salt substitutes may reduce the risk of recurrent stroke and death. In a recent study, compared to regular salt intake, salt substitutes reduced stroke-related mortality ...
The recommended daily amount of drinking water for humans varies. [1] It depends on activity, age, health, and environment.In the United States, the Adequate Intake for total water, based on median intakes, is 4.0 litres (141 imp fl oz; 135 US fl oz) per day for males older than 18, and 3.0 litres (106 imp fl oz; 101 US fl oz) per day for females over 18; it assumes about 80% from drink and 20 ...
Hyponatremia is caused by a relatively smaller loss in total body water. [19] Any cause of hypovolemia such as prolonged vomiting, decreased oral intake, severe diarrhea [12] Diuretic use (due to the diuretic causing a volume depleted state and thence ADH release, and not a direct result of diuretic-induced urine sodium loss) [12]