Search results
Results from the WOW.Com Content Network
Ammonium tetrathiomolybdate was first used therapeutically in the treatment of copper toxicosis in animals. It was then introduced as a treatment in Wilson's disease , a hereditary copper metabolism disorder, in humans; it acts both by competing with copper absorption in the bowel and by increasing excretion.
Molybdenum is an essential element in most organisms; a 2008 research paper speculated that a scarcity of molybdenum in the Earth's early oceans may have strongly influenced the evolution of eukaryotic life (which includes all plants and animals). [1] At least 50 molybdenum-containing enzymes have been identified, mostly in bacteria.
Molybdenum is also present within human tooth enamel and may help prevent its decay. [99] Acute toxicity has not been seen in humans, and the toxicity depends strongly on the chemical state. Studies on rats show a median lethal dose (LD 50) as low as 180 mg/kg for some Mo compounds. [100]
The agriculture industry uses 500 tons per year as a fertilizer. In particular, its use has been suggested for treatment of whiptail in broccoli and cauliflower in molybdenum-deficient soils. [4] [5] However, care must be taken because at a level of 0.3 ppm sodium molybdate can cause copper deficiencies in animals, particularly cattle. [3]
Molybdenum disulfide (or moly) is an inorganic compound composed of molybdenum and sulfur. Its chemical formula is MoS 2. The compound is classified as a transition metal dichalcogenide. It is a silvery black solid that occurs as the mineral molybdenite, the principal ore for molybdenum. [6] MoS 2 is relatively unreactive.
Molybdenum cofactor deficiency is a rare human disease in which the absence of molybdopterin – and consequently its molybdenum complex, commonly called molybdenum cofactor – leads to accumulation of toxic levels of sulphite and neurological damage.
Descriptions of human molybdenum deficiency are few. A patient receiving prolonged parenteral nutrition acquired a syndrome described as ‘acquired molybdenum deficiency.’ This syndrome, exacerbated by methionine administration, was characterized by high blood methionine, low blood uric acid , and low urinary uric acid and sulfate ...
The discrete molybdenum oxyanions range in size from the simplest MoO 2− 4, found in potassium molybdate up to extremely large structures found in isopoly-molybdenum blues that contain for example 154 Mo atoms. The behaviour of molybdenum is different from the other elements in group 6. Chromium only forms the chromates, CrO 2− 4, Cr 2 O 2 ...