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A pediatric concussion, also known as pediatric mild traumatic brain injury (mTBI), is a head trauma that impacts the brain capacity. Concussion can affect functional, emotional, cognitive and physical factors and can occur in people of all ages. [1]
Shaken baby syndrome (SBS), also known as abusive head trauma (AHT), is a controversial and scientifically disputed [4] [5] [6] medical condition in children younger than five years old, [3] hypothesized to be caused by blunt trauma, vigorous shaking, or a combination of both.
Complications from birth trauma can include damage to the head, spinal cord, soft tissues, and organs. [2]Trauma to the head of the infant can manifest as caput succedaneum, skull fractures, extracranial and intracranial hemorrhages, and cranial nerve injuries.
A concussion, also known as a mild traumatic brain injury (mTBI), is a head injury that temporarily affects brain functioning. [8] Symptoms may include headache, dizziness, difficulty with thinking and concentration, sleep disturbances, mood changes, a brief period of memory loss, brief loss of consciousness; problems with balance; nausea; blurred vision; and mood changes.
The subgaleal space is capable of holding up to 40% of a newborn baby's blood and can therefore result in acute shock and death. Fluid bolus may be required if blood loss is significant and the patient becomes tachycardic. Transfusion and phototherapy may be necessary. Investigation for coagulopathy may be indicated. [citation needed]
The fencing response is an unnatural position of the arms following a concussion.Immediately after moderate forces have been applied to the brainstem, the forearms are held flexed or extended (typically into the air) for a period lasting up to several seconds after the impact.
Instead, she uncovered a baby trafficking scandal in Georgia that has affected tens of thousands of lives. Over many decades, parents were lied to and told their newborn babies had died – the ...
Newborn hypoxic-ischaemic brain injury differs from injury in the adult brain in several ways: NMDA receptor toxicity is much higher in the immature brain. [18] Apoptotic mechanisms including activation of caspases, translocation of apoptosis -inducing factor and cytochrome-c release are much greater in the immature than the adult.