enow.com Web Search

Search results

  1. Results from the WOW.Com Content Network
  2. Vitamin K deficiency - Wikipedia

    en.wikipedia.org/wiki/Vitamin_K_deficiency

    Vitamin K 1-deficiency may occur by disturbed intestinal uptake (such as would occur in a bile duct obstruction), by therapeutic or accidental intake of a vitamin K 1-antagonist such as warfarin, or, very rarely, by nutritional vitamin K 1 deficiency. As a result, Gla-residues are inadequately formed and the Gla-proteins are insufficiently active.

  3. Vitamin K - Wikipedia

    en.wikipedia.org/wiki/Vitamin_K

    Vitamin K is a family of structurally similar, fat-soluble vitamers found in foods and marketed as dietary supplements. [1] The human body requires vitamin K for post-synthesis modification of certain proteins that are required for blood coagulation ("K" from Danish koagulation, for "coagulation") or for controlling binding of calcium in bones and other tissues. [2]

  4. Vitamin K reaction - Wikipedia

    en.wikipedia.org/wiki/Vitamin_K_reaction

    VKAs diminish vitamin K levels in the body and inhibit the synthesis of vitamin K dependent clotting factors. [27] Thus, by inhibiting vitamin K, a key element by which the body produces clots, the risk of prolonged bleeding increases. [28] Traditionally, vitamin K has been used as a reversal agent for VKAs.

  5. Madin-Darby canine kidney cells - Wikipedia

    en.wikipedia.org/wiki/Madin-Darby_Canine_Kidney...

    Following the initial isolation in 1958 of epithelial cells from the kidney tubule of an adult Cocker Spaniel dog by Stewart H. Madin and Norman B. Darby, Jr., [3] the cell line bearing their name was employed primarily as a model for viral infection of mammalian cells.

  6. VKORC1 - Wikipedia

    en.wikipedia.org/wiki/VKORC1

    The availability of reduced vitamin K is of importance for activation vitamin K 2,3-epoxide. The reduction of vitamin K epoxide is then responsible for the carboxylation of glutamic acid residues in some blood-clotting proteins, including factor VII, factor IX, and factor X. [5] [7] VKORC1 is of therapeutic interest both for its role in ...

  7. Dog health - Wikipedia

    en.wikipedia.org/wiki/Dog_health

    Then the dog is given Vitamin K supplementation for 3 to 4 weeks, depending on the type of poison. At the end of treatment, the clotting times should be tested again. The prognosis is good in these cases. However, if the dog is already showing signs of poisoning, it is too late to try to remove the poison from the body.

  8. Menatetrenone - Wikipedia

    en.wikipedia.org/wiki/Menatetrenone

    MK-4 is the major form of Vitamin K in vertebrate animals, including humans and common forms of meat animals. It is produced via conversion of vitamin K 1 in the body, specifically in the testes, pancreas and arterial walls. [2] The conversion is not dependent on gut bacteria, occurring in germ-free rats [3] [4] and in parenterally-administered ...

  9. Phytomenadione - Wikipedia

    en.wikipedia.org/wiki/Phytomenadione

    Phytomenadione, also known as vitamin K 1 or phylloquinone, is a vitamin found in food and used as a dietary supplement. [6] [7] It is on the World Health Organization's List of Essential Medicines. [8] It is used to treat certain bleeding disorders, [7] including warfarin overdose, vitamin K deficiency, and obstructive jaundice. [7]