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It has also been observed to cause villus ischemia, and increase intestinal motility. [15] The net result of these changes is induced diarrhoea. [15] Enteritis necroticans is an often fatal illness, caused by β-toxin of Clostridium perfringens. [16] This causes inflammation and segments of necrosis throughout the gastrointestinal tract.
Enteritis is the inflammation of the small intestine. It is generally caused by eating or drinking substances that are contaminated with bacteria or viruses. The bacterium and/or toxin settles in the small intestine and cause inflammation and swelling. This in turn can cause abdominal pain, cramping, diarrhea, fever, and dehydration. [1]
Gastroenteritis is defined as vomiting or diarrhea due to inflammation of the small or large bowel, often due to infection. [17] The changes in the small bowel are typically noninflammatory, while the ones in the large bowel are inflammatory. [17]
Enterocolitis is an inflammation of the digestive tract, involving enteritis of the small intestine and colitis of the colon. [1] It may be caused by various infections, with bacteria, viruses, fungi, parasites, or other causes.
Inflammatory bowel disease (IBD) is a group of inflammatory conditions of the colon and small intestine, with Crohn's disease and ulcerative colitis (UC) being the principal types. [3] Crohn's disease affects the small intestine and large intestine, as well as the mouth, esophagus, stomach and the anus, whereas UC primarily affects the colon ...
This can cause elevated physical temperature, painful spasms of the intestinal muscles , swelling due to fluid leaking from capillaries of the intestine and further tissue damage by the body's immune cells and the chemicals, called cytokines, which are released to fight the infection. The result can be impaired nutrient absorption, excessive ...
The organism produces diffuse, bloody, edematous, and exudative enteritis. In a small number of cases, the infection may be associated with hemolytic uremic syndrome and thrombotic thrombocytopenic purpura through a poorly understood mechanism. [citation needed]
A mutation in the cytosolic phospholipase A2-α gene has been identified as the cause of this disease in one family. [1] In this family the mutation was inherited as an autosomal recessive. It is not yet known if this gene is the cause of this disease in other families. The gene encoding cytosolic phospholipase A2-α is found on chromosome 1
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