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Lassa fever, also known as Lassa hemorrhagic fever, is a type of viral hemorrhagic fever caused by the Lassa virus. [1] Many of those infected by the virus do not develop symptoms . [ 1 ] When symptoms occur they typically include fever , weakness, headaches, vomiting , and muscle pains . [ 1 ]
Lassa virus life cycle. Figure by Fehling et al., 2012 [13] The life cycle of Lassa mammarenavirus is similar to the Old World arenaviruses. Lassa mammarenavirus enters the cell by the receptor-mediated endocytosis. Which endocytotic pathway is used is not known yet, but at least the cellular entry is sensitive to cholesterol depletion.
Viral replication is the formation of biological viruses during the infection process in the target host cells. Viruses must first get into the cell before viral replication can occur. Through the generation of abundant copies of its genome and packaging these copies, the virus continues infecting new hosts. Replication between viruses is ...
An arenavirus is a bi- or trisegmented ambisense RNA virus that is a member of the family Arenaviridae. [1] [2] These viruses infect rodents and occasionally humans.A class of novel, highly divergent arenaviruses, properly known as reptarenaviruses, have also been discovered which infect snakes to produce inclusion body disease, mostly in boa constrictors.
LCMV is a prototype of more severe hemorrhagic fever viruses, [8] especially Lassa virus with the greatest prevalence in sub-Saharan Africa. [8] However, other strains of this virus (Junin and Machupo viruses) [8] are present in parts of South America and other strains continue to significantly affect the southern African population. Since the ...
Bunyavirales is an order of segmented negative-strand RNA viruses with mainly tripartite genomes. Member viruses infect arthropods, plants, protozoans, and vertebrates. [2] It is the only order in the class Ellioviricetes. [1]
Replication cycle of vesicular stomatitis virus (VSV) Viral replication is cytoplasmic. The replication cycle is the same for most rhabdoviruses. All components required for early transcription and the nucleocapsid are released to the cytoplasm of the infected cell after the first steps of binding, penetration and uncoating take place. [9]
Transcription of mRNAs initiated by viral polymerase using cap snatching. The first step of transcription for some negative, single-stranded RNA viruses is cap snatching, in which the first 10 to 20 residues of a host cell RNA are removed (snatched) and used as the 5′ cap and primer to initiate the synthesis of the nascent viral mRNA. [1]
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