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Two giant platelets (stained purple) are visible in this image from a light microscope (40×) from a peripheral blood smear surrounded by red blood cells. One normal platelet can be seen in the upper left side of the image (purple) and is significantly smaller in size than the red blood cells (stained pink). Specialty: Hematology
Actin-binding proteins have been linked to maintaining the structure of the large platelets. However, enzymatic activity allows for functional clotting activity regardless of the size of the thrombocytes. [7] Genomic studies have found mutations in various pathways that could be the cause of Harris platelet syndrome, but further studies are ...
Reactive thrombocythemia is the most common cause of a high platelet count. It accounts for 88% to 97% of thrombocythemia cases in adults, and near 100% in children. In adults, acute infection, tissue damage, chronic inflammation and malignancy are the common causes of reactive thrombocythemia. Usually, one or more of these conditions is ...
In terms of diagnosis Bernard–Soulier syndrome is characterized by prolonged bleeding time, thrombocytopenia, increased megakaryocytes, and enlarged platelets, Bernard–Soulier syndrome is associated with quantitative or qualitative defects of the platelet glycoprotein complex GPIb/V/IX. The degree of thrombocytopenia may be estimated ...
[67] [68] Platelets were described in the early 19th century, and in the 1880s several investigators linked the purpura with abnormalities in the platelet count. [ 67 ] [ 69 ] The first report of a successful therapy for ITP was in 1916, when a young Polish medical student, Paul Kaznelson , described a female patient's response to a splenectomy ...
Increasing age, with the highest risk after ages 45 in men and 55 in women Being assigned male at birth Family history, especially having a genetic disorder called familial hypercholesterolemia
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GPS is characterized by "thrombocytopenia, abnormally large agranular platelets in peripheral blood smears, and almost total absence of platelet alpha-granules and their constituents." [4] The defect in GPS is the failure of megakaryocytes to package secretory proteins into alpha-granules. Patients with the GPS are affected by mild to moderate ...
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