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Critical autism studies (CAS) is an interdisciplinary research field within autism studies led by autistic people. [1] [2] [3] This field is related to both disability studies and neurodiversity studies. [4] [5] [6] CAS as a discipline is led by autistic academics, and many autistic people engage with the discipline in nonacademic spaces.
This perspective is distinct from two other likewise distinct views: the medical perspective, that autism is caused by a genetic defect and should be addressed by targeting the autism gene(s), and the fringe theory that autism is caused by environmental factors like vaccines and pollution and could be cured by addressing environmental causes. [75]
Models of disability are analytic tools in disability studies used to articulate different ways disability is conceptualized by individuals and society broadly. [1] [2] Disability models are useful for understanding disagreements over disability policy, [2] teaching people about ableism, [3] providing disability-responsive health care, [3] and articulating the life experiences of disabled people.
Neither category is satisfactory on its own; social cognition theories poorly address autism's rigid and repetitive behaviors, while most of the nonsocial theories have difficulty explaining social impairment and communication difficulties. [76] A combined theory based on multiple deficits may prove to be more useful. [77]
Autism spectrum disorder [a] (ASD), or simply autism, is a neurodevelopmental disorder "characterized by persistent deficits in social communication and social interaction across multiple contexts" and "restricted, repetitive patterns of behavior, interests, or activities". [11] Sensory abnormalities are also included in the diagnostic manuals ...
In 1964, Bernard Rimland published the book Infantile Autism: The Syndrome and Its Implications for a Neural Theory of Behavior, [177] [178] [179] which refuted the refrigerator theory. Instead, Rimland suggested, autism was a result of biochemical defects "triggered by environmental assaults". It included a foreword by Leo Kanner.
Opiate theory hypothesizes that autism is the result of a metabolic disorder in which opioid peptides gliadorphin (aka gluteomorphin) and Casomorphin, produced through metabolism of gluten (present in wheat and related cereals) and casein (present in dairy products), pass through an abnormally permeable intestinal wall and then proceed to exert ...
Whether autism has mainly a genetic or developmental cause, and the degree of coincidence between autism and intellectual disability, are all matters of current scientific controversy as well as inquiry. There is also more sociopolitical debate as to whether autism should be considered a disability on its own. [1]