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Neonatal jaundice is a yellowish discoloration of the white part of the eyes and skin in a newborn baby due to high bilirubin levels. [1] Other symptoms may include excess sleepiness or poor feeding. [ 1 ]
Physiologic jaundice can be a benign condition that presents in newborns until two weeks of life. [2] However, jaundice that continues after two weeks requires follow up with measurement of total and conjugated bilirubin. [3] Elevated levels of conjugated bilirubin are never benign and require further evaluation for neonatal cholestasis. [3]
In undiagnosed and untreated children, the accumulation of precursor metabolites due to the deficient activity of galactose 1-phosphate uridylyltransferase (GALT) can lead to feeding problems, failure to thrive, liver damage, bleeding, and infections. The first presenting symptom in an infant is often prolonged jaundice.
A Michigan couple’s newborn daughter died after they ignored a midwife’s warning that the baby’s jaundice could lead to brain damage or death.
The app could help identify severe cases in low and middle-income countries to prevent complications.
Most children born with congenital hypothyroidism and correctly treated with thyroxine grow and develop normally in all respects. Even most of those with athyreosis and undetectable T 4 levels at birth develop with normal intelligence, although as a population academic performance tends to be below that of siblings and mild learning problems ...
This allows a diagnosis to be made while the person is still an infant. Affected children can have serious, irreversible effects or even die within days from birth. Infants affected by galactosemia typically present with symptoms of lethargy, vomiting, diarrhea, failure to thrive, and jaundice.
Rainbow Babies: Stars Who Had Children After Miscarriages Read article “She developed a type of jaundice unique to her situation, and we transported [her] to the Children’s Hospital NICU in ...
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