Search results
Results from the WOW.Com Content Network
Developmental toxicity is any developmental malformation that is caused by the toxicity of a chemical or pathogen. It is the structural or functional alteration, reversible or irreversible, which interferes with homeostasis , normal growth , differentiation , development or behavior.
Environmental toxicants and fetal development is the impact of different toxic substances from the environment on the development of the fetus. This article deals with potential adverse effects of environmental toxicants on the prenatal development of both the embryo or fetus, as well as pregnancy complications. The human embryo or fetus is ...
Teratology is the study of abnormalities of physiological development in organisms during their life span. It is a sub-discipline in medical genetics which focuses on the classification of congenital abnormalities in dysmorphology caused by teratogens and also in pharmacology and toxicology.
Reproductive toxicity refers to the potential risk from a given chemical, physical or biologic agent to adversely affect both male and female fertility as well as offspring development. [1] Reproductive toxicants may adversely affect sexual function, ovarian failure, fertility as well as causing developmental toxicity in the offspring.
Category 1 has two subcategories for reproductive and developmental effects. Materials which cause concern for the health of breastfed children have a separate category: effects on or via Lactation. Specific target organ toxicity (STOT) [8] category distinguishes between single and repeated exposure for Target Organ Effects. All significant ...
Infants that are from pregnancy with a lot of stress are harder to soothe rather than those that mothers didn't experience any stress during pregnancy. Human and animal studies did show that infants from stressed pregnancies have a poor immune function and are more likely to contract childhood illnesses as well as mental disorders. [33]
Very few signs of toxicity are seen in adult animals after low doses, but developmental effects may occur at low dioxin levels, including foetal, neonatal, and possibly pubescent stages. [24] Well established developmental effects are cleft palate, hydronephrosis, disturbances in tooth development and sexual development, and endocrine effects. [24]
Long-term effects seem to include a slight excess of multiple myeloma and myeloid leukaemia, [19] as well as some developmental effects such as disturbed development of teeth [41] and excess of girls born to fathers who were exposed as children. [52] Several other long-term effects have been suspected, but the evidence is not very strong. [5]