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Acute inhalation injury may result from frequent and widespread use of household cleaning agents and industrial gases (including chlorine and ammonia).The airways and lungs receive continuous first-pass exposure to non-toxic and irritant or toxic gases via inhalation.
lung injury of acute onset, within 1 week of an apparent clinical insult and with the progression of respiratory symptoms; bilateral opacities on chest imaging (chest radiograph or CT) not explained by other lung pathology (e.g. effusion, lobar/lung collapse, or nodules) respiratory failure not explained by heart failure or volume overload ...
Smoke inhalation is the breathing in of harmful fumes (produced as by-products of combusting substances) through the respiratory tract. [1] This can cause smoke inhalation injury (subtype of acute inhalation injury) which is damage to the respiratory tract caused by chemical and/or heat exposure, as well as possible systemic toxicity after smoke inhalation.
There are two general types of chemical pneumonitis: acute and chronic. Irritants capable of causing chemical pneumonitis include vomitus , [ 2 ] barium used in gastro-intestinal imaging, chlorine gas (among other pulmonary agents ), [ 2 ] ingested gasoline [ 2 ] or other petroleum distillates , ingested or skin absorbed pesticides , [ 2 ...
Acute severe asthma, also known as status asthmaticus, is an acute exacerbation of asthma that does not respond to standard treatments of bronchodilators (inhalers) and corticosteroids. [2] Asthma is caused by multiple genes , some having protective effect, with each gene having its own tendency to be influenced by the environment although a ...
He spent 10 years as the frontman for the progressive rock band Indulge before starting a solo career. Tyler Golden/NBCU Photo Bank/NBCUniversal via Getty . Ryan Whyte Maloney on 'The Voice' in 2014.
Acute chlorine gas poisoning primarily affects the respiratory system, causing difficulty breathing, cough, irritation of the eyes, nose, and throat, and sometimes skin irritation. Higher exposures can lead to severe lung damage, such as toxic pneumonitis or pulmonary edema , with concentrations around 400 ppm and beyond potentially fatal.
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