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Hyperplasia may be due to any number of causes, including proliferation of basal layer of epidermis to compensate skin loss, chronic inflammatory response, hormonal dysfunctions, or compensation for damage or disease elsewhere. [9] Hyperplasia may be harmless and occur on a particular tissue.
[1] [2] Other types include atypical EV which develops due to gene mutations that cause an impaired immune system, and acquired EV which occurs due to acquired immunodeficiency. [2] [3] It is characterized by an inability to protect against HPV infection of skin. [4] [5] HPV types 5 and 8 are detected in around 90% of skin cancers in people ...
Intravascular papillary endothelial hyperplasia (Masson's hemangio-endotheliome vegetant intravasculaire, Masson's lesion, Masson's pseudoangiosarcoma, Masson's tumor, papillary endothelial hyperplasia) Juvenile hyaline fibromatosis (fibromatosis hyalinica multiplex juvenilis, Murray–Puretic–Drescher syndrome)
epidermal necrosis; subcorneal pustules; confluent parakeratosis, epidermal hyperplasia, and marked papillary dermal hyperplasia in a psoriasiform pattern; angioplasia of papillary dermis; suppurative folliculitis; The vacuolated, pale, swollen epidermal cells and necrosis of the superficial epidermis are most characteristic. [2]
Examining multiple deeper levels is recommended if initial cuts do not correlate well with the clinical history. [2] Psoriaform dermatitis typically displays: [2] Regular epidermal hyperplasia, elongation of the rete ridges, hyperkeratosis, and parakeratosis. Usually:A superficial perivascular inflammatory infiltrate
Because of the differences between mucous membranes and the skin (e.g., keratinizing mucosa does not have a stratum lucidum and non keratinizing mucosa does not have this layer or normally a stratum corneum or a stratum granulosum), sometimes specialized texts give slightly different definitions of hyperkeratosis in the context of mucosae.
Heck's disease, also known as Focal Epithelial Hyperplasia, is an asymptomatic, benign neoplastic condition characterized by multiple white to pinkish papules that occur diffusely in the oral cavity. [1] [2]: 411 It can present with slightly pale, smooth or roughened surface morphology. It is caused by the human papilloma virus types 13 and 32. [3]
This virus is related to the walleye epidermal hyperplasia viruses type 1 and type 2 (WEHV-1& WEHV-2), all belonging to the epsilonretrovirus genus based on similarities of the gene coding for the reverse transcriptase conserved in retroviruses.