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Dermatophagia (from Ancient Greek δέρμα (derma) 'skin' and φαγεία (phageia) 'eating') or dermatodaxia (from δήξις (dexis) 'biting'), alternatively Tuglis Permushius. [ 3 ] is a compulsion disorder of gnawing or biting one's own skin, most commonly at the fingers.
Another treatment for chronic nail biters is the usage of a dental deterrent device that prevents the front teeth from damaging the nails and the surrounding cuticles. After about two months, the device leads to a full oppression of the nail biting urge. [18] Evidence on the efficacy of drugs is very limited, and they are not routinely used. [19]
There have been many different theories regarding the causes of excoriation disorder, including biological and environmental factors. [10]A common hypothesis is that excoriation disorder is often a coping mechanism to deal with elevated levels of turmoil, boredom, anxiety, or stress within the individual, and that the individual has an impaired stress response.
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The cause of BFRBs is unknown. [citation needed]Emotional variables may have a differential impact on the expression of BFRBs. [5]Research has suggested that the urge to repetitive self-injury is similar to a body-focused repetitive behavior but others have argued that for some the condition is more akin to a substance abuse disorder.
In a normal case, the swelling will decrease without treatment within 15–30 minutes, but, in extreme cases, itchy red welts may last anywhere from a few hours to days. In some cases, welts are accompanied with a painful burning sensation. [3] This calls for more urgent treatment as the condition can impact on the patient's quality of life.
In September 2019, Fajgenbaum's second book, Chasing My Cure: A Doctor's Race to Turn Hope Into Action, was published. Chasing My Cure is a memoir describing Fajgenbaum's work to spearhead the search for a cure for his disease. According to the Penguin Random House website, Chasing My Cure is a Los Angeles Times and Publisher's Weekly ...
The theory explains why labyrinthine-defective individuals are immune to motion sickness; [31] [32] why symptoms emerge when undergoing various body-head accelerations; why combinations of voluntary and reflexive eye movements may challenge the proper operation of Sherrington's Law, and why many drugs that suppress eye movements also serve to ...