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Pennisetum alopecuroides (Pen-ih-SEE-tum al-oh-pek-yur-OY-deez), [1] the Chinese pennisetum, Chinese fountaingrass, dwarf fountain grass, foxtail fountain grass, or swamp foxtail grass, is a species of perennial grass native to Asia and Australia. The culms are erect, and 60–100 cm long.
Pennisetum is closely related to the genus Cenchrus, [11] and the boundary between them is unclear. [12] Cenchrus was derived from Pennisetum and the two are grouped in a monophyletic clade. [13] Some species now in Pennisetum were once members of Cenchrus, and some have been moved back.
APS-2 (Schmidt's disease): Autoimmune Addison's disease (constantly present), in conjunction with either type 1 diabetes mellitus (DM) or autoimmune thyroid disease. [4] APS-3: Type 1 diabetes, atrophic gastritis, pernicious anemia, vitiligo, alopecia, and myasthenia gravis, and autoimmune thyroid disease; Addison's disease and/or ...
Cenchrus is a widespread genus of plants in the grass family. [3] [6] Its species are native to many countries in Asia, Africa, Australia, the Americas, and various oceanic islands.
A diagnosis of CUP requires a clinical picture consistent with metastatic disease and one or more biopsy results inconsistent with a tumor cancer. CUP is found in about 3 to 5% of all people diagnosed with invasive cancer, [1] and carries a poor prognosis in most (80 to 85%) of those circumstances. The other 15 to 20% of patients, however, have ...
CLOVES syndrome is a rare overgrowth syndrome with complex vascular anomalies. CLOVES syndrome affects people with various symptoms, ranging from mild fatty soft-tissue tumors to vascular malformations encompassing the spine or internal organs.
Retinal vasculopathy with cerebral leukocencephalopathy and systemic manifestations (RVCL or RVCL-S, also previously known as retinal vasculopathy with cerebral leukodystrophy, RVCL; or cerebroretinal vasculopathy, CRV; or hereditary vascular retinopathy, HVR; or hereditary endotheliopathy, retinopathy, nephropathy, and stroke, HERNS) is an inherited condition resulting from a frameshift ...
In terms of the diagnosis of adenylosuccinate lyase deficiency one should look for (or exam/method): [4] [3] Demonstration of succinylpurines in extracellular fluids like plasma, cerebrospinal fluid and/or urine using high-pressure liquid chromatography, with or without mass spectroscopy