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Instead, he suggests testing sleep onset latency during the day, ideally at 10:00 a.m., 12:30 p.m. and 3:00 p.m. A sleep onset latency of 0 to 5 minutes indicates severe sleep deprivation, 5 to 10 minutes is "troublesome", 10 to 15 minutes indicates a mild but "manageable" degree of sleep debt, and 15 to 20 minutes is indicative of "little or ...
Polysomnography data can be directly related to sleep onset latency (SOL), REM-sleep onset latency, number of awakenings during the sleep period, total sleep duration, percentages and durations of every sleep stage, and number of arousals. It may also record other information crucial for diagnostics that are not directly linked with sleep, such ...
The Multiple Sleep Latency Test (MSLT) is a sleep disorder diagnostic tool. It is used to measure the time elapsed from the start of a daytime nap period to the first signs of sleep, called sleep latency. The test is based on the idea that the sleepier people are, the faster they will fall asleep.
REM-on neurons are primarily cholinergic (i.e., involve acetylcholine); REM-off neurons activate serotonin and noradrenaline, which among other functions suppress the REM-on neurons. McCarley and Hobson suggested that the REM-on neurons actually stimulate REM-off neurons, thereby serving as the mechanism for the cycling between REM and non-REM ...
An indicator of sleep propensity can also be seen in the shortening of the transition from light stages of non-REM sleep to deeper slow-wave oscillations. [69] On average, the latency in healthy adults decreases by a few minutes after a night without sleep, and the latency from sleep onset to slow-wave sleep is halved. [69]
SWS is potentially decreased (sometimes totally absent), spindles and the time spent in REM sleep are also reduced, while its latency is increased. [168] The poorly sleep onset in AD has been also associated with dream-related hallucination, increased restlessness, wandering and agitation, that seem to be related with sundowning - a typical ...
Sleep onset is the transition from wakefulness into sleep. Sleep onset usually transits into non-rapid eye movement sleep (NREM sleep) but under certain circumstances (e.g. narcolepsy ) it is possible to transit from wakefulness directly into rapid eye movement sleep (REM sleep).
REM sleep is decreased during the first half of the sleep period and stage 1 sleep is increased in the second half of the sleep period. [5] Most antidepressants, in particular selective serotonin re-uptake inhibitors (SSRIs), such as citalopram and paroxetine, are potent inhibitors of REM sleep and may also cause a REM rebound on discontinuation.