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Chlamydia psittaci is a lethal intracellular bacterial species that may cause endemic avian chlamydiosis, epizootic outbreaks in other mammals, and respiratory psittacosis in humans. Potential hosts include feral birds and domesticated poultry, as well as cattle, pigs, sheep, and horses. C. psittaci is transmitted by inhalation, contact, or ...
Psittacosis—also known as parrot fever, and ornithosis—is a zoonotic infectious disease in humans caused by a bacterium called Chlamydia psittaci and contracted from infected parrots, such as macaws, cockatiels, and budgerigars, and from pigeons, sparrows, ducks, hens, gulls and many other species of birds.
Bird flu is a scary illness with a high mortality rate. But so far, infections in the U.S. have been relatively mild—until now. A patient in Louisiana has been hospitalized with a severe case of ...
The virus Psittacid alphaherpesvirus 1 is the etiologic agent that causes Pacheco's disease. This virus species is closely related to Gallid alphaherpesvirus 1. [2] It was initially identified as a herpesvirus by examining its virion size, sensitivity to ether, the formation of intranuclear inclusions, its ability to thicken the nuclear membranes of the host cells.
Humans infected with bird flu display a wide range of symptoms, from pneumonia requiring hospitalization to no symptoms at all. The CDC lists these other possible signs of infection : Cough
What are the symptoms of bird flu in humans? Reported signs and symptoms of bird flu virus infections in humans have ranged from no symptoms, mild, to severe illness. Symptoms include:
Bird flu can also cause respiratory and classic flu-like symptoms, including cough, runny nose, fever, sore throat, body aches, headaches, fatigue, shortness of breath, and pneumonia, the CDC says ...
Chlamydia abortus is a species in Chlamydiota that causes abortion and fetal death in mammals, including humans. Chlamydia abortus was renamed in 1999 as Chlamydophila psittaci along with all Chlamydiota except Chlamydia trachomatis. This was based on a lack of evident glycogen production and on resistance to the antibiotic sulfadiazine.