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These molecular abnormalities damage the cerebellum and hippocampus, which results in deficiencies in long-term memory functions and spatial working memory. Chronic sleep deprivation also raises the brain's amyloid-beta aggregation, which is linked to neurodegenerative diseases like Alzheimer's.
The hippocampus is located in the medial temporal lobe (subcortical), and is an infolding of the medial temporal cortex. [1] The hippocampus plays an important role in the transfer of information from short-term memory to long-term memory during encoding and retrieval stages. These stages do not need to occur successively, but are, as studies ...
In a study done by Mark A. Smith, it is demonstrated that exposure to continuous stress can cause age-related issues to the hippocampus. [27] What then becomes more noticeable is that the aging brain is not as able to recognize growth, this is a symptom of hippocampal damage. If the information is not being encoded properly in the brain then of ...
Studies on specific cases demonstrate how particular impaired areas of the hippocampus are associated with the severity of RA. Damage can be limited to the CA1 field of the hippocampus, causing very limited RA for about one to two years. [13] More extensive damage limited to the hippocampus causes temporally graded amnesia for 15 to 25 years. [13]
Alcohol acts as a general central nervous system depressant, but it also affects some specific areas of the brain to a greater extent than others. Memory impairment caused by alcohol has been linked to the disruption of hippocampal function—particularly affecting gamma-Aminobutyric acid (GABA) and N-methyl-D-aspartate (NMDA) neurotransmission which negatively impacts long-term potentiation ...
In Alzheimer's disease (and other forms of dementia), the hippocampus is one of the first regions of the brain to suffer damage; short-term memory loss and disorientation are included among the early symptoms. Damage to the hippocampus can also result from oxygen starvation , encephalitis, or medial temporal lobe epilepsy.
The hippocampus is the brain region located in the medial temporal lobe, responsible for forming new episodic and semantic memories. As a result of his neurological damage, Cochrane suffered severe cognitive deficits that hindered his ability to form new episodic memories. However, both his semantic memory and noetic consciousness remained ...
Hippocampal sclerosis occurs with severe CA1 and less severe CA3 and CA4 neuronal loss. [27] Experimental research has shown that N-methyl-d-aspartate (NMDA) receptor activation causes neuronal cell loss, and electrical stimulation-induced animal models of temporal lobe epilepsy duplicate the cell loss pattern of temporal lobe epilepsy in ...