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Gum disease is typically caused by a bacterial infection that results from an overgrowth of dental plaque — a sticky, colorless film — on the teeth and gums.
Gingivitis is a non-destructive disease that causes inflammation of the gums; [1] ulitis is an alternative term. [2] The most common form of gingivitis, and the most common form of periodontal disease overall, is in response to bacterial biofilms (also called plaque) that are attached to tooth surfaces, termed plaque-induced gingivitis.
Periodontal disease; Other names: Gum disease, pyorrhea, periodontitis: Radiograph showing bone loss between the two roots of a tooth (black region). The spongy bone has receded due to infection under tooth, reducing the bony support for the tooth.
Necrotizing gingivitis (NG) is a common, non-contagious infection of the gums with sudden onset.The main features are painful, bleeding gums, and ulceration of interdental papillae (the sections of gum between adjacent teeth).
Chronic periodontitis is initiated by Gram-negative tooth-associated microbial biofilms that elicit a host response, which results in bone and soft tissue destruction. In response to endotoxin derived from periodontal pathogens, several osteoclast-related mediators target the destruction of alveolar bone and supporting connective tissue such as the periodontal ligament.
Necrotizing periodontal diseases are non-contagious infections but may occasionally occur in epidemic-like patterns due to shared risk factors. The milder form, necrotizing ulcerative gingivitis (also termed "trench mouth"), [9] is characterized by painful, bleeding gums and ulceration and necrosis of the interdental papilla.
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