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The main treatment for it is avoiding contact with nickel-releasing metals, such as inexpensive jewelry. Another form of nickel allergy is a systemic form: systemic nickel allergy syndrome (SNAS) can mimic some of the symptoms of irritable bowel syndrome (IBS) and also has a dermatologic component. [1]
Nickel is the most common contact allergen worldwide (of people with contact dermatitis, 11.4% in Europe, 8.8–25.7% in China, and 17.5% in North America are allergic to nickel). [1] Nickel allergy, and contact allergies more generally, can develop when people are any age, but they are most likely to develop in early adulthood.
An elimination diet, also known as exclusion diet, is a diagnostic procedure used to identify foods that an individual cannot consume without adverse effects. [1] Adverse effects may be due to food allergy , food intolerance , other physiological mechanisms (such as metabolic or toxins), [ 2 ] or a combination of these.
A patch test (contact delayed hypersensitivity allergy test) [17] is a commonly used examination to determine the exact cause of an allergic contact dermatitis. According to the American Academy of Allergy, Asthma, and Immunology, "patch testing is the gold standard for contact allergen identification". [2]
Nevertheless, the term Nickel Directive is still used to refer to the restrictions on nickel usage and the prescribed test method for quantifying nickel release from products EN 1811. Allergy to nickel is a common cause of contact dermatitis , with roughly 10% of the population in Western Europe and North America being sensitive to nickel.
Common causes of allergic contact dermatitis include: nickel allergy, 14K or 18K gold, Balsam of Peru (Myroxylon pereirae), and chromium. In the Americas they include the oily, urushiol-containing coating from plants of the genus Toxicodendron: poison ivy, poison oak, and poison sumac. Millions of cases occur each year in North America alone. [10]
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Enzyme potentiated desensitization (EPD), is a treatment for allergies developed in the 1960s by Dr. Leonard M. McEwen in the United Kingdom.EPD uses much lower doses of antigens than conventional desensitization treatment paired with the enzyme β-glucuronidase.
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