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Skull bossing is a descriptive term in medical physical examination indicating a protuberance of the skull, most often in the frontal bones of the forehead ("frontal bossing"). Although prominence of the skull bones may be normal, skull bossing may be associated with certain medical conditions, [ 1 ] including nutritional, metabolic, hormonal ...
Compensatory growth occurs forward at the coronal suture and backward at the lambdoid suture giving respectively a prominent forehead, called frontal bossing, and a prominent back portion of the head, called coning. [10] [11] This is the most common form of craniosynostosis. [13]
Parrot's sign, [3] also known as 'Parrot's nodes' [4] and 'Parrot's bosses', [5] [6] refers to the bony growth noted at autopsy by Marie Jules Parrot and Jonathan Hutchinson on the skulls of children with congenital syphilis (CS) in the 19th century. [2] [7] Later publications also describe it as the frontal bossing that presents in the late ...
Cranial sutures. A defining characteristic of Crouzon syndrome is craniosynostosis, which results in an abnormal head shape.This is present in combinations of: frontal bossing, trigonocephaly (fusion of the metopic suture), brachycephaly (fusion of the coronal suture), dolichocephaly (fusion of the sagittal suture), plagiocephaly (unilateral premature closure of lambdoid and coronal sutures ...
Acute periostitis is due to infection, characterized by diffuse formation of pus, severe pain, and constitutional symptoms, and usually results in necrosis.It can be caused by excessive physical activity as well, as in the case of medial tibial stress syndrome (also referred to as tibial periostalgia, soleus periostalgia, or shin splints).
Paget's disease affecting the skull may cause frontal bossing, increased hat size, and headaches. Often patients may develop loss of hearing in one or both ears [7] due to auditory foramen narrowing and resultant compression of the nerves in the inner ear.
The disease was first described by Maroteaux and Lamy in 1962 [4] [5] at which time it was defined by the following characteristics: dwarfism; osteopetrosis; partial agenesis of the terminal digits of the hands and feet; cranial anomalies, such as persistence of fontanelles and failure of closure of cranial sutures; frontal and occipital bossing; and hypoplasia of the angle of the mandible. [6]
This disease affects more men than women. [1] [4] After onset, the disease stabilizes after about 5–20 years. Life of PDP patients can be severely impaired. [1] [5] Currently, symptomatic treatments are NSAIDs and steroids or surgical procedures. [5] [6] In 1868, PDP was first described by Friedreich as 'excessive growth of bone of the entire ...