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Not only that, they had a 52 percent lower risk of vascular dementia, and a 39 percent lower risk of Alzheimer’s dementia. People who took SGLT-2 inhibitors for longer periods of time seemed to ...
Dementia prevention is a critical area of research, as experts want to understand what people can do to decrease dementia risk. A recent study found that more than 5 years of taking medications ...
Vascular dementia can sometimes be triggered by cerebral amyloid angiopathy, which involves accumulation of amyloid beta plaques in the walls of the cerebral arteries, leading to breakdown and rupture of the vessels. [2] [5] Since amyloid plaques are a characteristic feature of Alzheimer's disease, vascular dementia may occur as a consequence ...
More must be done to see whether existing drugs could help to tackle dementia, scientists have said after finding that a number of common treatments may affect a person’s risk.
Antihypertensive and antidiabetic medications in individuals without overt cognitive impairment may decrease the risk of dementia by influencing cerebrovascular pathology. [ 1 ] [ 167 ] More research is needed to examine the relationship with AD specifically; clarification of the direct role medications play versus other concurrent lifestyle ...
The prevention of dementia involves reducing the number of risk factors for the development of dementia, and is a global health priority needing a global response. [1] [2] Initiatives include the establishment of the International Research Network on Dementia Prevention (IRNDP) [3] which aims to link researchers in this field globally, and the establishment of the Global Dementia Observatory ...
Heart problems can increase dementia risk, but a new study suggests that heparin, a common anticoagulant administered via injection, may help delay Alzheimer’s onset.
[8] [12] Memantine was first studied in the treatment of Alzheimer's disease in 1986. [13] [14] The drug was first marketed for dementia in 1989 in Germany under the name Axura. [8] [14] [12] It was not discovered to act as an NMDA receptor antagonist until 1989, after clinical trials had initiated.
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