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  2. ADAM17 - Wikipedia

    en.wikipedia.org/wiki/ADAM17

    A disintegrin and metalloprotease 17 (ADAM17), also called TACE (tumor necrosis factor-α-converting enzyme), is a 70-kDa enzyme that belongs to the ADAM protein family of disintegrins and metalloproteases, activated by substrate presentation.

  3. ADAM (protein) - Wikipedia

    en.wikipedia.org/wiki/ADAM_(protein)

    ADAMs (short for a disintegrin and metalloproteinase) are a family of single-pass transmembrane and secreted metalloendopeptidases. [1] [2] All ADAMs are characterized by a particular domain organization featuring a pro-domain, a metalloprotease, a disintegrin, a cysteine-rich, an epidermal-growth factor like and a transmembrane domain, as well as a C-terminal cytoplasmic tail. [3]

  4. List of primary immunodeficiencies - Wikipedia

    en.wikipedia.org/wiki/List_of_primary_immuno...

    Hyper-IgD syndrome (Mevalonate kinase deficiency) CIAS1-related diseases: Muckle–Wells syndrome; Familial cold autoinflammatory syndrome, types 1, 2, 3, and 4; Neonatal onset multisystem inflammatory disease; NLRP1 deficiency; PAPA syndrome (pyogenic sterile arthritis, pyoderma gangrenosum, acne) ADAM17 deficiency; Blau syndrome

  5. Isolated 17,20-lyase deficiency - Wikipedia

    en.wikipedia.org/.../Isolated_17,20-lyase_deficiency

    The symptoms of isolated 17,20-lyase deficiency, in males, include pseudohermaphroditism (i.e., feminized, ambiguous, or mildly underdeveloped (e.g., micropenis, perineal hypospadias, and/or cryptorchidism (undescended testes)) external genitalia), female gender identity, and, in non-complete cases of deficiency where partial virilization occurs, gynecomastia up to Tanner stage V (due to low ...

  6. Cytochrome P450 oxidoreductase deficiency - Wikipedia

    en.wikipedia.org/wiki/Cytochrome_P450...

    Symptoms of severe forms of PORD include ambiguous genitalia in males and females, congenital adrenal hyperplasia, cortisol deficiency, and Antley–Bixler skeletal malformation syndrome (ABS), while symptoms of mild forms include polycystic ovary syndrome in women and hypogonadism in men. [3]

  7. Congenital adrenal hyperplasia due to 17α-hydroxylase deficiency

    en.wikipedia.org/wiki/Congenital_adrenal...

    The sex steroid deficiency produces effects similar to 17α-hydroxylase deficiency. Severely affected genetic females (XX) are born with normal internal and external genitalia and there are no clues to abnormality until adolescence, when the androgenic and estrogenic signs (e.g., breasts and pubic hair) of puberty either fails to occur or is ...

  8. Nutritionists Explain Whether Women Over 50 Can Try ... - AOL

    www.aol.com/nutritionists-explain-whether-women...

    How much protein do women over 50 need? If you’re on an intermittent fasting diet, it’s important to focus on good nutrition during your eating windows, Cording says. That means focusing on a ...

  9. Congenital adrenal hyperplasia due to 21-hydroxylase deficiency

    en.wikipedia.org/wiki/Congenital_adrenal...

    Women with classical CAH have statistically reduced fertility, especially those with the salt-losing form. [75] Live birth rate is 33–50% in simple virilized form of CAH and 0–10% in most severe salt-wasting form. In the nonclassical form of CAH, the live birth is 63–90%, similar to the age-matched control groups. [76]